Overexpression of BDNF and Full-Length TrkB Receptor Ameliorate Striatal Neural Survival in Huntington's Disease.

Silva, Ana; Naia, Luana; Dominguez, Alejandro; Ribeiro, Márcio; Rodrigues, Joana; Vieira, Otília V; Lessmann, Volkmar; Rego, Ana Cristina

Silva, Ana; Naia, Luana; Dominguez, Alejandro; Ribeiro, Márcio; Rodrigues, Joana; Vieira, Otília V; Lessmann, Volkmar; Rego, Ana Cristina.

Affiliation

Silva A; CNC - Center for Neuroscience and Cell Biology, University of Coimbra, Coimbra, Portugal.

Neurodegener Dis ; 15(4): 207-18, 2015.

Article in En | MEDLINE | ID: mdl-25896770

Subject(s)

Apoptosis; Brain-Derived Neurotrophic Factor/metabolism; Corpus Striatum/metabolism; Huntington Disease/metabolism; Membrane Glycoproteins/metabolism; Neurons/metabolism; Protein-Tyrosine Kinases/metabolism; Caspase 3/metabolism; Cell Line; Humans; Huntingtin Protein; MAP Kinase Signaling System; Nerve Tissue Proteins/metabolism; Proto-Oncogene Proteins c-akt/metabolism; Receptor, trkB; Recombinant Proteins/metabolism; Signal Transduction

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Full text: 1 Database: MEDLINE Main subject: Protein-Tyrosine Kinases / Membrane Glycoproteins / Apoptosis / Huntington Disease / Brain-Derived Neurotrophic Factor / Corpus Striatum / Neurons Limits: Humans Language: En Journal: Neurodegener Dis Journal subject: NEUROLOGIA Year: 2015 Type: Article Affiliation country: Portugal

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