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Schnyder corneal dystrophy-associated UBIAD1 is defective in MK-4 synthesis and resists autophagy-mediated degradation.
Jun, Dong-Jae; Schumacher, Marc M; Hwang, Seonghwan; Kinch, Lisa N; Grishin, Nick V; DeBose-Boyd, Russell A.
Affiliation
  • Jun DJ; Departments of Molecular Genetics,University of Texas Southwestern Medical Center, Dallas, TX 75390-9046.
  • Schumacher MM; Departments of Molecular Genetics,University of Texas Southwestern Medical Center, Dallas, TX 75390-9046.
  • Hwang S; Departments of Molecular Genetics,University of Texas Southwestern Medical Center, Dallas, TX 75390-9046.
  • Kinch LN; Biophysics,University of Texas Southwestern Medical Center, Dallas, TX 75390-9046.
  • Grishin NV; Biophysics,University of Texas Southwestern Medical Center, Dallas, TX 75390-9046; Howard Hughes Medical Institute,University of Texas Southwestern Medical Center, Dallas, TX 75390-9046.
  • DeBose-Boyd RA; Departments of Molecular Genetics,University of Texas Southwestern Medical Center, Dallas, TX 75390-9046. Electronic address: mailto:Russell.DeBose-Boyd@utsouthwestern.edu.
J Lipid Res ; 61(5): 746-757, 2020 05.
Article in En | MEDLINE | ID: mdl-32188638
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Full text: 1 Database: MEDLINE Main subject: Autophagy / Genetic Variation / Corneal Dystrophies, Hereditary / Vitamin K 2 / Dimethylallyltranstransferase / Proteolysis Type of study: Risk_factors_studies Limits: Humans Language: En Journal: J Lipid Res Year: 2020 Type: Article
Fulltext
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Full text: 1 Database: MEDLINE Main subject: Autophagy / Genetic Variation / Corneal Dystrophies, Hereditary / Vitamin K 2 / Dimethylallyltranstransferase / Proteolysis Type of study: Risk_factors_studies Limits: Humans Language: En Journal: J Lipid Res Year: 2020 Type: Article