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A low amyloidogenic E61K transthyretin mutation may cause familial amyloid polyneuropathy.
Murakami, Tatsufumi; Yokoyama, Takeshi; Mizuguchi, Mineyuki; Toné, Shigenobu; Takaku, Shizuka; Sango, Kazunori; Nishimura, Hirotake; Watabe, Kazuhiko; Sunada, Yoshihide.
Affiliation
  • Murakami T; Department of Neurology, Kawasaki Medical School, Kurashiki, Japan.
  • Yokoyama T; Faculty of Pharmaceutical Sciences, University of Toyama, Toyama, Japan.
  • Mizuguchi M; Faculty of Pharmaceutical Sciences, University of Toyama, Toyama, Japan.
  • Toné S; Graduate School of Science and Engineering, Tokyo Denki University, Hatoyama, Saitama, Japan.
  • Takaku S; Diabetic Neuropathy Project, Department of Diseases and Infection, Tokyo Metropolitan Institute of Medical Science, Tokyo, Japan.
  • Sango K; Diabetic Neuropathy Project, Department of Diseases and Infection, Tokyo Metropolitan Institute of Medical Science, Tokyo, Japan.
  • Nishimura H; Department of Pathology, Kawasaki Medical School, Kurashiki, Japan.
  • Watabe K; Department of Medical Technology, Faculty of Health Sciences, Kyorin University, Tokyo, Japan.
  • Sunada Y; Department of Neurology, Kawasaki Medical School, Kurashiki, Japan.
J Neurochem ; 156(6): 957-966, 2021 03.
Article in En | MEDLINE | ID: mdl-32852783
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Full text: 1 Database: MEDLINE Main subject: Prealbumin / Amyloid Neuropathies, Familial / Amyloid Limits: Animals / Humans Language: En Journal: J Neurochem Year: 2021 Type: Article Affiliation country: Japan
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Full text: 1 Database: MEDLINE Main subject: Prealbumin / Amyloid Neuropathies, Familial / Amyloid Limits: Animals / Humans Language: En Journal: J Neurochem Year: 2021 Type: Article Affiliation country: Japan