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Modeling Potential Autophagy Pathways in COVID-19 and Sarcoidosis.
Calender, Alain; Israel-Biet, Dominique; Valeyre, Dominique; Pacheco, Yves.
Affiliation
  • Calender A; Department of Genetics, University Hospital, University Claude Bernard Lyon 1, Lyon, France. Electronic address: alain.calender@chu-lyon.fr.
  • Israel-Biet D; Department of Pulmonology AP-HP, Georges Pompidou European Hospital, Paris, France.
  • Valeyre D; Department of Pulmonology, Avicenne Hospital, Bobigny, France; Saint Joseph Hospital, APHP, Paris, France; University Sorbonne Paris Nord, INSERM UMR 1272, Villetaneuse, France.
  • Pacheco Y; Department of Genetics, University Hospital, University Claude Bernard Lyon 1, Lyon, France.
Trends Immunol ; 41(10): 856-859, 2020 10.
Article in En | MEDLINE | ID: mdl-32863134
Coronavirus disease 2019 (COVID-19) is caused by severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) and mainly affects the lungs. Sarcoidosis is an autoinflammatory disease characterized by the diffusion of granulomas in the lungs and other organs. Here, we discuss how the two diseases might involve some common mechanistic cellular pathways around the regulation of autophagy.
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Full text: 1 Database: MEDLINE Main subject: Pneumonia, Viral / Pulmonary Edema / Sarcoidosis / Autophagy / Coronavirus Infections / Severe Acute Respiratory Syndrome / Betacoronavirus Language: En Journal: Trends Immunol Journal subject: ALERGIA E IMUNOLOGIA Year: 2020 Type: Article
Fulltext
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Full text: 1 Database: MEDLINE Main subject: Pneumonia, Viral / Pulmonary Edema / Sarcoidosis / Autophagy / Coronavirus Infections / Severe Acute Respiratory Syndrome / Betacoronavirus Language: En Journal: Trends Immunol Journal subject: ALERGIA E IMUNOLOGIA Year: 2020 Type: Article