Forsythiaside A Alleviates Lipopolysaccharide-Induced Acute Liver Injury through Inhibiting Endoplasmic Reticulum Stress and NLRP3 Inflammasome Activation.

Fu, Jing-Nan; Liu, Shu-Chang; Chen, Yi; Zhao, Jie; Lu, Ning; Ma, Tao

Fu, Jing-Nan; Liu, Shu-Chang; Chen, Yi; Zhao, Jie; Lu, Ning; Ma, Tao.

Affiliation

Fu JN; Department of General Surgery, Tianjin Medical University General Hospital.
Liu SC; Department of Minimally Invasive Surgery, Characteristics Medical Center of Chinese People Armed Police Force.
Chen Y; Department of General Surgery, Tianjin Medical University General Hospital.
Zhao J; Department of General Surgery, Tianjin Medical University General Hospital.
Lu N; Department of Intensive Care Unit, Tianjin Medical University General Hospital.
Ma T; Department of General Surgery, Tianjin Medical University General Hospital.

Biol Pharm Bull ; 46(7): 979-986, 2023 Jul 01.

Article in En | MEDLINE | ID: mdl-37183023

Subject(s)

Inflammasomes; Sepsis; Mice; Animals; Inflammasomes/metabolism; NLR Family, Pyrin Domain-Containing 3 Protein/metabolism; Lipopolysaccharides/pharmacology; Liver/metabolism; Tumor Necrosis Factor-alpha/metabolism; Endoplasmic Reticulum Stress; Sepsis/pathology

Key words

endoplasmic reticulum stress; forsythiaside A; lipopolysaccharide; liver injury; mouse; nucleotide-binding oligomerization domain-like receptor pyrin domain containing 3 (NLRP3) inflammasome

Fulltext

Add to My VHL

XML

PubMed Links

Search on Google

Full text: 1 Database: MEDLINE Main subject: Sepsis / Inflammasomes Limits: Animals Language: En Journal: Biol Pharm Bull Journal subject: BIOQUIMICA / FARMACOLOGIA Year: 2023 Type: Article

Fulltext

Add to My VHL

XML

PubMed Links

Search on Google