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Disruption of neuronal RHEB signaling impairs oligodendrocyte differentiation and myelination through mTORC1-DLK1 axis.
Huang, Haijiao; Jing, Bo; Zhu, Feiyan; Jiang, Wanxiang; Tang, Ping; Shi, Liyang; Chen, Huiting; Ren, Guoru; Xia, Shiyao; Wang, Luoling; Cui, Yiyuan; Yang, Zhiwen; Platero, Alexander J; Hutchins, Andrew P; Chen, Mina; Worley, Paul F; Xiao, Bo.
Affiliation
  • Huang H; Departments of Neuroscience and Systems Biology, School of Life Sciences, Southern University of Science and Technology, Shenzhen Key Laboratory for Gene Regulation and Systems Biology, Shenzhen 518055, People's Republic of China.
  • Jing B; Departments of Neuroscience and Systems Biology, School of Life Sciences, Southern University of Science and Technology, Shenzhen Key Laboratory for Gene Regulation and Systems Biology, Shenzhen 518055, People's Republic of China. Electronic address: jing_scenery@126.com.
  • Zhu F; Departments of Neuroscience and Systems Biology, School of Life Sciences, Southern University of Science and Technology, Shenzhen Key Laboratory for Gene Regulation and Systems Biology, Shenzhen 518055, People's Republic of China.
  • Jiang W; State Key Laboratory of Biotherapy, West China Hospital, Sichuan University, Chengdu 610041, People's Republic of China.
  • Tang P; State Key Laboratory of Biotherapy, West China Hospital, Sichuan University, Chengdu 610041, People's Republic of China.
  • Shi L; Departments of Neuroscience and Systems Biology, School of Life Sciences, Southern University of Science and Technology, Shenzhen Key Laboratory for Gene Regulation and Systems Biology, Shenzhen 518055, People's Republic of China.
  • Chen H; Departments of Neuroscience and Systems Biology, School of Life Sciences, Southern University of Science and Technology, Shenzhen Key Laboratory for Gene Regulation and Systems Biology, Shenzhen 518055, People's Republic of China.
  • Ren G; Departments of Neuroscience and Systems Biology, School of Life Sciences, Southern University of Science and Technology, Shenzhen Key Laboratory for Gene Regulation and Systems Biology, Shenzhen 518055, People's Republic of China.
  • Xia S; Departments of Neuroscience and Systems Biology, School of Life Sciences, Southern University of Science and Technology, Shenzhen Key Laboratory for Gene Regulation and Systems Biology, Shenzhen 518055, People's Republic of China.
  • Wang L; Departments of Neuroscience and Systems Biology, School of Life Sciences, Southern University of Science and Technology, Shenzhen Key Laboratory for Gene Regulation and Systems Biology, Shenzhen 518055, People's Republic of China.
  • Cui Y; State Key Laboratory of Biotherapy, West China Hospital, Sichuan University, Chengdu 610041, People's Republic of China.
  • Yang Z; Departments of Neuroscience and Systems Biology, School of Life Sciences, Southern University of Science and Technology, Shenzhen Key Laboratory for Gene Regulation and Systems Biology, Shenzhen 518055, People's Republic of China.
  • Platero AJ; The Solomon H. Snyder Department of Neuroscience, Johns Hopkins University School of Medicine, Baltimore, MD 21205, USA.
  • Hutchins AP; Departments of Neuroscience and Systems Biology, School of Life Sciences, Southern University of Science and Technology, Shenzhen Key Laboratory for Gene Regulation and Systems Biology, Shenzhen 518055, People's Republic of China.
  • Chen M; State Key Laboratory of Biotherapy, West China Hospital, Sichuan University, Chengdu 610041, People's Republic of China.
  • Worley PF; The Solomon H. Snyder Department of Neuroscience, Johns Hopkins University School of Medicine, Baltimore, MD 21205, USA. Electronic address: pworley1@jhmi.edu.
  • Xiao B; Departments of Neuroscience and Systems Biology, School of Life Sciences, Southern University of Science and Technology, Shenzhen Key Laboratory for Gene Regulation and Systems Biology, Shenzhen 518055, People's Republic of China. Electronic address: xiaob@sustech.edu.cn.
Cell Rep ; 42(7): 112801, 2023 07 25.
Article in En | MEDLINE | ID: mdl-37463107
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Full text: 1 Database: MEDLINE Main subject: Signal Transduction / Ras Homolog Enriched in Brain Protein / Myelin Sheath Limits: Animals Language: En Journal: Cell Rep Year: 2023 Type: Article
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Full text: 1 Database: MEDLINE Main subject: Signal Transduction / Ras Homolog Enriched in Brain Protein / Myelin Sheath Limits: Animals Language: En Journal: Cell Rep Year: 2023 Type: Article