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HER2 genomic amplification in circulating tumor DNA from patients with cetuximab-resistant colorectal cancer.
Takegawa, Naoki; Yonesaka, Kimio; Sakai, Kazuko; Ueda, Hiroto; Watanabe, Satomi; Nonagase, Yoshikane; Okuno, Tatsuya; Takeda, Masayuki; Maenishi, Osamu; Tsurutani, Junji; Satoh, Taroh; Okamoto, Isamu; Nishio, Kazuto; Tamura, Takao; Nakagawa, Kazuhiko.
Afiliación
  • Takegawa N; Department of Medical Oncology, Kinki University School of Medicine, Osaka, Japan.
  • Yonesaka K; Department of Medical Oncology, Kinki University School of Medicine, Osaka, Japan.
  • Sakai K; Department of Genome Biology, Kinki University School of Medicine, Osaka, Japan.
  • Ueda H; Department of Medical Oncology, Kinki University School of Medicine, Osaka, Japan.
  • Watanabe S; Department of Medical Oncology, Kinki University School of Medicine, Osaka, Japan.
  • Nonagase Y; Department of Medical Oncology, Kinki University School of Medicine, Osaka, Japan.
  • Okuno T; Department of Medical Oncology, Kinki University School of Medicine, Osaka, Japan.
  • Takeda M; Department of Medical Oncology, Kinki University School of Medicine, Osaka, Japan.
  • Maenishi O; Department of Pathology, Kinki University School of Medicine, Osaka, Japan.
  • Tsurutani J; Department of Medical Oncology, Kinki University School of Medicine, Osaka, Japan.
  • Satoh T; Department of Frontier Science for Cancer and Chemotherapy, Osaka University Graduate School of Medicine, Osaka, Japan.
  • Okamoto I; Center for Clinical and Translational Research, Kyushu University, Kyushu, Japan.
  • Nishio K; Department of Genome Biology, Kinki University School of Medicine, Osaka, Japan.
  • Tamura T; Department of Medical Oncology, Kinki University School of Medicine, Osaka, Japan.
  • Nakagawa K; Department of Medical Oncology, Kinki University School of Medicine, Osaka, Japan.
Oncotarget ; 7(3): 3453-60, 2016 Jan 19.
Article en En | MEDLINE | ID: mdl-26657506
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Texto completo: 1 Bases de datos: MEDLINE Asunto principal: ADN de Neoplasias / Neoplasias Colorrectales / Amplificación de Genes / Receptor ErbB-2 / Resistencia a Antineoplásicos / Cetuximab / Células Neoplásicas Circulantes Tipo de estudio: Etiology_studies / Incidence_studies / Observational_studies / Prognostic_studies / Risk_factors_studies Límite: Aged / Aged80 / Female / Humans / Male / Middle aged Idioma: En Revista: Oncotarget Año: 2016 Tipo del documento: Article País de afiliación: Japón
Texto completo
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Texto completo: 1 Bases de datos: MEDLINE Asunto principal: ADN de Neoplasias / Neoplasias Colorrectales / Amplificación de Genes / Receptor ErbB-2 / Resistencia a Antineoplásicos / Cetuximab / Células Neoplásicas Circulantes Tipo de estudio: Etiology_studies / Incidence_studies / Observational_studies / Prognostic_studies / Risk_factors_studies Límite: Aged / Aged80 / Female / Humans / Male / Middle aged Idioma: En Revista: Oncotarget Año: 2016 Tipo del documento: Article País de afiliación: Japón