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Mitophagy in Cardiomyocytes and in Platelets: A Major Mechanism of Cardioprotection Against Ischemia/Reperfusion Injury.
Zhang, Weilin; Chen, Chuyan; Wang, Jun; Liu, Lei; He, Yubin; Chen, Quan.
Afiliación
  • Zhang W; The State Key Laboratory of Membrane Biology, Institute of Zoology, Chinese Academy of Sciences , Beijing , China.
  • Chen C; Chinese Academy of Medical Sciences and School of Basic Medicine, Peking Union Medical College , Beijing , China.
  • Wang J; The State Key Laboratory of Membrane Biology, Institute of Zoology, Chinese Academy of Sciences , Beijing , China.
  • Liu L; The State Key Laboratory of Membrane Biology, Institute of Zoology, Chinese Academy of Sciences , Beijing , China.
  • He Y; Department of Cardiology, Heart Center, Chinese Army General Hospital , Beijing , China.
  • Chen Q; The State Key Laboratory of Membrane Biology, Institute of Zoology, Chinese Academy of Sciences , Beijing , China.
Physiology (Bethesda) ; 33(2): 86-98, 2018 03 01.
Article en En | MEDLINE | ID: mdl-29412057
ABSTRACT
Mitophagy, a process that selectively removes damaged organelles by autolysosomal degradation, is an early cellular response to ischemia. Mitophagy is activated in both cardiomyocytes and platelets during ischemia/reperfusion (I/R) and heart disease conditions. We focus on the molecular regulation of mitophagy and highlight the role of mitophagy in cardioprotection.
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Texto completo: 1 Bases de datos: MEDLINE Asunto principal: Plaquetas / Daño por Reperfusión / Miocitos Cardíacos / Mitofagia / Mitocondrias Límite: Animals / Humans Idioma: En Revista: Physiology (Bethesda) Asunto de la revista: FISIOLOGIA Año: 2018 Tipo del documento: Article País de afiliación: China
Texto completo
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Texto completo: 1 Bases de datos: MEDLINE Asunto principal: Plaquetas / Daño por Reperfusión / Miocitos Cardíacos / Mitofagia / Mitocondrias Límite: Animals / Humans Idioma: En Revista: Physiology (Bethesda) Asunto de la revista: FISIOLOGIA Año: 2018 Tipo del documento: Article País de afiliación: China