Competition for shared downstream signaling molecules establishes indirect negative feedback between EGFR and EphA2.

Oh, Dongmyung; Chen, Zhongwen; Biswas, Kabir H; Bai, Funing; Ong, Hui Ting; Sheetz, Michael P; Groves, Jay T

Oh, Dongmyung; Chen, Zhongwen; Biswas, Kabir H; Bai, Funing; Ong, Hui Ting; Sheetz, Michael P; Groves, Jay T.

Afiliación

Oh D; Mechanobiology Institute, National University of Singapore, Singapore, Singapore; Department of Biochemistry and Molecular Biology, The University of Texas Medical Branch, Galveston, Texas.
Chen Z; Mechanobiology Institute, National University of Singapore, Singapore, Singapore; Interdisciplinary Research Center on Biology and Chemistry, Shanghai Institute of Organic Chemistry, Chinese Academy of Science, Shanghai, China.
Biswas KH; Mechanobiology Institute, National University of Singapore, Singapore, Singapore; Division of Biological and Biomedical Sciences, College of Health & Life Sciences, Hamad Bin Khalifa University, Qatar Foundation, Doha, Qatar.
Bai F; Mechanobiology Institute, National University of Singapore, Singapore, Singapore.
Ong HT; Mechanobiology Institute, National University of Singapore, Singapore, Singapore.
Sheetz MP; Mechanobiology Institute, National University of Singapore, Singapore, Singapore; Department of Biochemistry and Molecular Biology, The University of Texas Medical Branch, Galveston, Texas. Electronic address: misheetz@utmb.edu.
Groves JT; Department of Chemistry, University of California, Berkeley, California; Institute for Digital Molecular Analytics and Science, Nanyang Technological University, Singapore, Singapore. Electronic address: jtgroves@lbl.gov.

Biophys J ; 121(10): 1897-1908, 2022 05 17.

Article en En | MEDLINE | ID: mdl-35430415

Asunto(s)

Efrina-A1; Receptor EphA2; Factor de Crecimiento Epidérmico; Receptores ErbB/metabolismo; Retroalimentación; Receptor EphA2/metabolismo; Transducción de Señal

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Texto completo: 1 Bases de datos: MEDLINE Asunto principal: Receptor EphA2 / Efrina-A1 Idioma: En Revista: Biophys J Año: 2022 Tipo del documento: Article