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Cell-Autonomous Constitutive gp130 Signaling in T Cells Amplifies TH17 Cell Responses and Causes Severe Lung Inflammation.
Heinig, Lisa Charlotte; Huth, Emily Valentina Madelaine; Yan, Karsten; Schumacher, Neele; Nawrocki, Mikolaj; Lory, Niels Christian; Bradtke, Peter; Bertram, Tabea; Rattay, Guido; Schmid, Joanna; Huber, Samuel; Wiech, Thorsten; Schmidt-Arras, Dirk; Rose-John, Stefan; Mittrücker, Hans-Willi.
Afiliación
  • Heinig LC; Institute for Immunology, University Medical Center Hamburg-Eppendorf, Hamburg, Germany.
  • Huth EVM; Institute for Immunology, University Medical Center Hamburg-Eppendorf, Hamburg, Germany.
  • Yan K; Institute for Immunology, University Medical Center Hamburg-Eppendorf, Hamburg, Germany.
  • Schumacher N; Institute of Biochemistry, Christian Albrechts University Kiel, Kiel, Germany.
  • Nawrocki M; I. Department of Medicine, University Medical Center Hamburg-Eppendorf, Hamburg, Germany.
  • Lory NC; Institute for Immunology, University Medical Center Hamburg-Eppendorf, Hamburg, Germany.
  • Bradtke P; Institute for Immunology, University Medical Center Hamburg-Eppendorf, Hamburg, Germany.
  • Bertram T; Institute for Immunology, University Medical Center Hamburg-Eppendorf, Hamburg, Germany.
  • Rattay G; I. Department of Medicine, University Medical Center Hamburg-Eppendorf, Hamburg, Germany.
  • Schmid J; Institute for Immunology, University Medical Center Hamburg-Eppendorf, Hamburg, Germany.
  • Huber S; I. Department of Medicine, University Medical Center Hamburg-Eppendorf, Hamburg, Germany.
  • Wiech T; Hamburg Center for Translational Immunology, University Medical Center Hamburg-Eppendorf, Hamburg, Germany.
  • Schmidt-Arras D; Institute for Pathology, University Medical Center Hamburg-Eppendorf, Hamburg, Germany.
  • Rose-John S; Institute of Biochemistry, Christian Albrechts University Kiel, Kiel, Germany.
  • Mittrücker HW; Department of Biosciences and Medical Biology, Paris Lodron University Salzburg, Salzburg, Austria.
J Immunol ; 210(11): 1717-1727, 2023 06 01.
Article en En | MEDLINE | ID: mdl-37058116
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Texto completo: 1 Bases de datos: MEDLINE Asunto principal: Neumonía / Células Th17 Tipo de estudio: Etiology_studies Límite: Animals Idioma: En Revista: J Immunol Año: 2023 Tipo del documento: Article País de afiliación: Alemania
Texto completo
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XML
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Texto completo: 1 Bases de datos: MEDLINE Asunto principal: Neumonía / Células Th17 Tipo de estudio: Etiology_studies Límite: Animals Idioma: En Revista: J Immunol Año: 2023 Tipo del documento: Article País de afiliación: Alemania