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NRIP1/RIP140 siRNA-mediated attenuation counteracts mitochondrial dysfunction in Down syndrome.
Izzo, Antonella; Manco, Rosanna; Bonfiglio, Ferdinando; Calì, Gaetano; De Cristofaro, Tiziana; Patergnani, Simone; Cicatiello, Rita; Scrima, Rosella; Zannini, Mariastella; Pinton, Paolo; Conti, Anna; Nitsch, Lucio.
Afiliação
  • Izzo A; Department of Molecular Medicine and Medical Biotechnology, University of Naples Federico II, Via Pansini 5, Naples 80131, Italy.
  • Manco R; Department of Molecular Medicine and Medical Biotechnology, University of Naples Federico II, Via Pansini 5, Naples 80131, Italy.
  • Bonfiglio F; Department of Molecular Medicine and Medical Biotechnology, University of Naples Federico II, Via Pansini 5, Naples 80131, Italy.
  • Calì G; Institute of Experimental Endocrinology and Oncology, National Research Council, Naples 80131, Italy.
  • De Cristofaro T; Institute of Experimental Endocrinology and Oncology, National Research Council, Naples 80131, Italy.
  • Patergnani S; Department of Experimental and Diagnostic Medicine, University of Ferrara, Ferrara 44100, Italy.
  • Cicatiello R; Department of Molecular Medicine and Medical Biotechnology, University of Naples Federico II, Via Pansini 5, Naples 80131, Italy.
  • Scrima R; Department of Clinical and Experimental Medicine, University of Foggia, Foggia 71100, Italy.
  • Zannini M; Institute of Experimental Endocrinology and Oncology, National Research Council, Naples 80131, Italy.
  • Pinton P; Department of Experimental and Diagnostic Medicine, University of Ferrara, Ferrara 44100, Italy.
  • Conti A; Department of Molecular Medicine and Medical Biotechnology, University of Naples Federico II, Via Pansini 5, Naples 80131, Italy anconti@unina.it.
  • Nitsch L; Department of Molecular Medicine and Medical Biotechnology, University of Naples Federico II, Via Pansini 5, Naples 80131, Italy.
Hum Mol Genet ; 23(16): 4406-19, 2014 Aug 15.
Article em En | MEDLINE | ID: mdl-24698981

Texto completo: 1 Bases de dados: MEDLINE Assunto principal: Trissomia / Cromossomos Humanos Par 21 / Proteínas Nucleares / Síndrome de Down / Proteínas Adaptadoras de Transdução de Sinal / Mitocôndrias / Miocárdio Tipo de estudo: Systematic_reviews Limite: Humans Idioma: En Revista: Hum Mol Genet Assunto da revista: BIOLOGIA MOLECULAR / GENETICA MEDICA Ano de publicação: 2014 Tipo de documento: Article País de afiliação: Itália

Texto completo: 1 Bases de dados: MEDLINE Assunto principal: Trissomia / Cromossomos Humanos Par 21 / Proteínas Nucleares / Síndrome de Down / Proteínas Adaptadoras de Transdução de Sinal / Mitocôndrias / Miocárdio Tipo de estudo: Systematic_reviews Limite: Humans Idioma: En Revista: Hum Mol Genet Assunto da revista: BIOLOGIA MOLECULAR / GENETICA MEDICA Ano de publicação: 2014 Tipo de documento: Article País de afiliação: Itália