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Egr1 confers protection against acetaminophen­induced hepatotoxicity via transcriptional upregulating of Acaa2.
Lei, Xiaohong; Xu, Qingling; Li, Chunmin; Niu, Baolin; Ming, Yanan; Li, Jing; Tang, Yingyue; Li, Xiaoyun; Tang, Jieting; Wu, Jing; Ju, Yi; Yao, Lvfeng; Wang, Bin; Miao, Qi; Zhong, Wei; Zhi, Yang; Xu, Lirong; Li, Chaojun; Li, Xiaobo; Mao, Yimin.
Afiliação
  • Lei X; Division of Gastroenterology and Hepatology; Shanghai Institute of Digestive Disease; NHC Key Laboratory of Digestive Diseases; Renji Hospital, School of Medicine, Shanghai Jiao Tong University, Shanghai, 200001, China.
  • Xu Q; Department of Hepatology, Mengchao Hepatobiliary Hospital of Fujian Medical University, Fuzhou, Fujian, 350025, China.
  • Li C; Division of Gastroenterology and Hepatology; Shanghai Institute of Digestive Disease; NHC Key Laboratory of Digestive Diseases; Renji Hospital, School of Medicine, Shanghai Jiao Tong University, Shanghai, 200001, China.
  • Niu B; Department of Gastroenterology and Hepatology, Shanghai General Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, 200080, China.
  • Ming Y; Department of Physiology and Pathophysiology, School of Basic Medical Sciences, Fudan University, Shanghai, 200032, China.
  • Li J; Division of Gastroenterology and Hepatology; Shanghai Institute of Digestive Disease; NHC Key Laboratory of Digestive Diseases; Renji Hospital, School of Medicine, Shanghai Jiao Tong University, Shanghai, 200001, China.
  • Tang Y; Division of Gastroenterology and Hepatology; Shanghai Institute of Digestive Disease; NHC Key Laboratory of Digestive Diseases; Renji Hospital, School of Medicine, Shanghai Jiao Tong University, Shanghai, 200001, China.
  • Li X; Division of Gastroenterology and Hepatology; Shanghai Institute of Digestive Disease; NHC Key Laboratory of Digestive Diseases; Renji Hospital, School of Medicine, Shanghai Jiao Tong University, Shanghai, 200001, China.
  • Tang J; Institute of Pathology, University Hospital Heidelberg, Heidelberg, 69120, Germany.
  • Wu J; Division of Gastroenterology and Hepatology; Shanghai Institute of Digestive Disease; NHC Key Laboratory of Digestive Diseases; Renji Hospital, School of Medicine, Shanghai Jiao Tong University, Shanghai, 200001, China.
  • Ju Y; Division of Gastroenterology and Hepatology; Shanghai Institute of Digestive Disease; NHC Key Laboratory of Digestive Diseases; Renji Hospital, School of Medicine, Shanghai Jiao Tong University, Shanghai, 200001, China.
  • Yao L; Model Animal Research Centre (MARC), Medical School of Nanjing University, National Resource Centre for Mutant Mice, Nanjing, 210093, China.
  • Wang B; Department of Physiology and Pathophysiology, School of Basic Medical Sciences, Fudan University, Shanghai, 200032, China.
  • Miao Q; Department of Hepatology, Mengchao Hepatobiliary Hospital of Fujian Medical University, Fuzhou, Fujian, 350025, China.
  • Zhong W; Department of Hepatology, Mengchao Hepatobiliary Hospital of Fujian Medical University, Fuzhou, Fujian, 350025, China.
  • Zhi Y; Department of Pathology, School of Basic Medical Sciences, Fujian Medical University, Fuzhou, Fujian, 350122, China.
  • Xu L; Division of Gastroenterology and Hepatology; Shanghai Institute of Digestive Disease; NHC Key Laboratory of Digestive Diseases; Renji Hospital, School of Medicine, Shanghai Jiao Tong University, Shanghai, 200001, China.
  • Li C; Division of Gastroenterology and Hepatology; Shanghai Institute of Digestive Disease; NHC Key Laboratory of Digestive Diseases; Renji Hospital, School of Medicine, Shanghai Jiao Tong University, Shanghai, 200001, China.
  • Li X; Division of Gastroenterology and Hepatology; Shanghai Institute of Digestive Disease; NHC Key Laboratory of Digestive Diseases; Renji Hospital, School of Medicine, Shanghai Jiao Tong University, Shanghai, 200001, China.
  • Mao Y; Department of Physiology and Pathophysiology, School of Basic Medical Sciences, Fudan University, Shanghai, 200032, China.
Int J Biol Sci ; 18(9): 3800-3817, 2022.
Article em En | MEDLINE | ID: mdl-35813467
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Texto completo: 1 Bases de dados: MEDLINE Assunto principal: Doença Hepática Induzida por Substâncias e Drogas / Acetaminofen Tipo de estudo: Prognostic_studies Limite: Animals Idioma: En Revista: Int J Biol Sci Assunto da revista: BIOLOGIA Ano de publicação: 2022 Tipo de documento: Article País de afiliação: China
Texto completo
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Texto completo: 1 Bases de dados: MEDLINE Assunto principal: Doença Hepática Induzida por Substâncias e Drogas / Acetaminofen Tipo de estudo: Prognostic_studies Limite: Animals Idioma: En Revista: Int J Biol Sci Assunto da revista: BIOLOGIA Ano de publicação: 2022 Tipo de documento: Article País de afiliação: China