Detalhe da pesquisa
1.
Implication of satellite cell behaviors in capillary growth via VEGF expression-independent mechanism in response to mechanical loading in HeyL-null mice.
Am J Physiol Cell Physiol
; 322(2): C275-C282, 2022 02 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-35020502
2.
Cell-autonomous and redundant roles of Hey1 and HeyL in muscle stem cells: HeyL requires Hes1 to bind diverse DNA sites.
Development
; 146(4)2019 02 20.
Artigo
em Inglês
| MEDLINE | ID: mdl-30745427
3.
The SGLT2 Inhibitor Luseogliflozin Rapidly Normalizes Aortic mRNA Levels of Inflammation-Related but Not Lipid-Metabolism-Related Genes and Suppresses Atherosclerosis in Diabetic ApoE KO Mice.
Int J Mol Sci
; 18(8)2017 Aug 04.
Artigo
em Inglês
| MEDLINE | ID: mdl-28777298
4.
Ectopic retinoic acid signaling affects outflow tract cushion development through suppression of the myocardial Tbx2-Tgfß2 pathway.
Development
; 139(2): 385-95, 2012 Jan.
Artigo
em Inglês
| MEDLINE | ID: mdl-22186728
5.
Hesr1 and Hesr3 are essential to generate undifferentiated quiescent satellite cells and to maintain satellite cell numbers.
Development
; 138(21): 4609-19, 2011 Nov.
Artigo
em Inglês
| MEDLINE | ID: mdl-21989910
6.
Mastermind-like 1 (MamL1) and mastermind-like 3 (MamL3) are essential for Notch signaling in vivo.
Development
; 138(23): 5235-46, 2011 Dec.
Artigo
em Inglês
| MEDLINE | ID: mdl-22069191
7.
Enhanced prepulse inhibition and low sensitivity to a dopamine agonist in HESR1 knockout mice.
J Neurosci Res
; 92(3): 287-97, 2014 Mar.
Artigo
em Inglês
| MEDLINE | ID: mdl-24431082
8.
Hesr2 knockout mice develop aortic valve disease with advancing age.
Arterioscler Thromb Vasc Biol
; 33(3): e84-92, 2013 Mar.
Artigo
em Inglês
| MEDLINE | ID: mdl-23288164
9.
Endothelin receptor type A expression defines a distinct cardiac subdomain within the heart field and is later implicated in chamber myocardium formation.
Development
; 137(22): 3823-33, 2010 Nov.
Artigo
em Inglês
| MEDLINE | ID: mdl-20929948
10.
Induction of Timp1 in smooth muscle cells during development of abdominal aortic aneurysms.
Hiroshima J Med Sci
; 62(3): 63-7, 2013 Sep.
Artigo
em Inglês
| MEDLINE | ID: mdl-24279124
11.
Angiotensin II Type 1 Receptor Blocker Prevents Abdominal Aortic Aneurysm Progression in Osteoprotegerin-Deficient Mice via Upregulation of Angiotensin (1-7).
J Am Heart Assoc
; 12(3): e027589, 2023 02 07.
Artigo
em Inglês
| MEDLINE | ID: mdl-36718875
12.
Sox9 Inhibits Cochlear Hair Cell Fate by Upregulating Hey1 and HeyL Antagonists of Atoh1.
Cells
; 12(17)2023 08 25.
Artigo
em Inglês
| MEDLINE | ID: mdl-37681879
13.
S-1-Propenylcysteine promotes IL-10-induced M2c macrophage polarization through prolonged activation of IL-10R/STAT3 signaling.
Sci Rep
; 11(1): 22469, 2021 11 17.
Artigo
em Inglês
| MEDLINE | ID: mdl-34789834
14.
Smad4 regulates the nuclear translocation of Nkx2-5 in cardiac differentiation.
Sci Rep
; 11(1): 3588, 2021 02 11.
Artigo
em Inglês
| MEDLINE | ID: mdl-33574455
15.
Disruption of Osteoprotegerin has complex effects on medial destruction and adventitial fibrosis during mouse abdominal aortic aneurysm formation.
PLoS One
; 15(7): e0235553, 2020.
Artigo
em Inglês
| MEDLINE | ID: mdl-32614927
16.
Hesr1 and Hesr2 may act as early effectors of Notch signaling in the developing cochlea.
Dev Biol
; 316(1): 87-99, 2008 Apr 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-18291358
17.
Sustained expression of HeyL is critical for the proliferation of muscle stem cells in overloaded muscle.
Elife
; 82019 09 23.
Artigo
em Inglês
| MEDLINE | ID: mdl-31545169
18.
Sfrp5 identifies murine cardiac progenitors for all myocardial structures except for the right ventricle.
Nat Commun
; 8: 14664, 2017 03 13.
Artigo
em Inglês
| MEDLINE | ID: mdl-28287088
19.
Targeted disruption of hesr2 results in atrioventricular valve anomalies that lead to heart dysfunction.
Circ Res
; 95(5): 540-7, 2004 Sep 03.
Artigo
em Inglês
| MEDLINE | ID: mdl-15297376
20.
Hesr, a mediator of the Notch signaling, functions in heart and vessel development.
Trends Cardiovasc Med
; 15(5): 190-4, 2005 Jul.
Artigo
em Inglês
| MEDLINE | ID: mdl-16165016