Detalhe da pesquisa
1.
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2.
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3.
Publisher Correction: Clonal analysis of Salmonella-specific effector T cells reveals serovar-specific and cross-reactive T cell responses.
Nat Immunol
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em Inglês
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4.
Clonal analysis of Salmonella-specific effector T cells reveals serovar-specific and cross-reactive T cell responses.
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5.
An HLA-E-targeted TCR bispecific molecule redirects T cell immunity against Mycobacterium tuberculosis.
Proc Natl Acad Sci U S A
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em Inglês
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Unconventional MAIT cell responses to bacterial infections.
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The P5-type ATPase ATP13A1 modulates major histocompatibility complex I-related protein 1 (MR1)-mediated antigen presentation.
J Biol Chem
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Ligand-dependent downregulation of MR1 cell surface expression.
Proc Natl Acad Sci U S A
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Evasion of MAIT cell recognition by the African Salmonella Typhimurium ST313 pathovar that causes invasive disease.
Proc Natl Acad Sci U S A
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10.
Invariant NKT cells modulate the suppressive activity of IL-10-secreting neutrophils differentiated with serum amyloid A.
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Sterile activation of invariant natural killer T cells by ER-stressed antigen-presenting cells.
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Deletion of the deISGylating enzyme USP18 enhances tumour cell antigenicity and radiosensitivity.
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Activation of Human Mucosal-Associated Invariant T Cells Induces CD40L-Dependent Maturation of Monocyte-Derived and Primary Dendritic Cells.
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14.
The actin cytoskeleton modulates the activation of iNKT cells by segregating CD1d nanoclusters on antigen-presenting cells.
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Elevated and cross-responsive CD1a-reactive T cells in bee and wasp venom allergic individuals.
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NKT-dependent B-cell activation in Gaucher disease.
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17.
Essential role for autophagy during invariant NKT cell development.
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18.
Cross-reactivity of hepatitis C virus specific vaccine-induced T cells at immunodominant epitopes.
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19.
Saposins modulate human invariant Natural Killer T cells self-reactivity and facilitate lipid exchange with CD1d molecules during antigen presentation.
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20.
Globosides but not isoglobosides can impact the development of invariant NKT cells and their interaction with dendritic cells.
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