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MicroRNA-340 as a modulator of RAS-RAF-MAPK signaling in melanoma.
Poenitzsch Strong, Ashley M; Setaluri, Vijayasaradhi; Spiegelman, Vladimir S.
Afiliação
  • Poenitzsch Strong AM; Department of Dermatology, University of Wisconsin-Madison, School of Medicine and Public Health, 1300 University Avenue, Madison, WI 53706, United States; Molecular and Environmental Toxicology Center, University of Wisconsin-Madison, School of Medicine and Public Health, 1300 University Avenue, Madison, WI 53706, United States.
  • Setaluri V; Department of Dermatology, University of Wisconsin-Madison, School of Medicine and Public Health, 1300 University Avenue, Madison, WI 53706, United States; Molecular and Environmental Toxicology Center, University of Wisconsin-Madison, School of Medicine and Public Health, 1300 University Avenue, Madison, WI 53706, United States; UW Carbone Cancer Center, University of Wisconsin-Madison, School of Medicine and Public Health, 600 Highland Avenue, Madison, WI 53792, United States.
  • Spiegelman VS; Department of Dermatology, University of Wisconsin-Madison, School of Medicine and Public Health, 1300 University Avenue, Madison, WI 53706, United States; Molecular and Environmental Toxicology Center, University of Wisconsin-Madison, School of Medicine and Public Health, 1300 University Avenue, Madison, WI 53706, United States; UW Carbone Cancer Center, University of Wisconsin-Madison, School of Medicine and Public Health, 600 Highland Avenue, Madison, WI 53792, United States. Electronic addre
Arch Biochem Biophys ; 563: 118-24, 2014 Dec 01.
Article em En | MEDLINE | ID: mdl-25043973

Texto completo: 1 Base de dados: MEDLINE Assunto principal: Proteínas ras / Sistema de Sinalização das MAP Quinases / MicroRNAs / Quinases raf / Melanoma Tipo de estudo: Etiology_studies Limite: Humans Idioma: En Ano de publicação: 2014 Tipo de documento: Article

Texto completo: 1 Base de dados: MEDLINE Assunto principal: Proteínas ras / Sistema de Sinalização das MAP Quinases / MicroRNAs / Quinases raf / Melanoma Tipo de estudo: Etiology_studies Limite: Humans Idioma: En Ano de publicação: 2014 Tipo de documento: Article