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Soluble prion protein and its N-terminal fragment prevent impairment of synaptic plasticity by Aß oligomers: Implications for novel therapeutic strategy in Alzheimer's disease.
Scott-McKean, Jonah J; Surewicz, Krystyna; Choi, Jin-Kyu; Ruffin, Vernon A; Salameh, Ahlam I; Nieznanski, Krzysztof; Costa, Alberto C S; Surewicz, Witold K.
Afiliação
  • Scott-McKean JJ; Division of Pediatric Neurology, Department of Pediatrics, Cleveland, OH, USA 44116.
  • Surewicz K; Department of Physiology and Biophysics, Cleveland, OH, USA 44116.
  • Choi JK; Department of Physiology and Biophysics, Cleveland, OH, USA 44116.
  • Ruffin VA; Department of Physiology and Biophysics, Cleveland, OH, USA 44116.
  • Salameh AI; Department of Physiology and Biophysics, Cleveland, OH, USA 44116.
  • Nieznanski K; Department of Biochemistry, Nencki Institute of Experimental Biology, 02-093Warsaw, Poland.
  • Costa ACS; Division of Pediatric Neurology, Department of Pediatrics, Cleveland, OH, USA 44116.
  • Surewicz WK; Department of Psychiatry, Case Western Reserve University, Cleveland, OH, USA 44116.
Neurobiol Dis ; 91: 124-131, 2016 07.
Article em En | MEDLINE | ID: mdl-26949218
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Texto completo: 1 Base de dados: MEDLINE Assunto principal: Peptídeos beta-Amiloides / Potenciação de Longa Duração / Doença de Alzheimer / Proteínas Priônicas / Plasticidade Neuronal Limite: Animals / Humans Idioma: En Ano de publicação: 2016 Tipo de documento: Article
Texto completo
Imprimir
XML
PubMed Links
Buscar no Google

Texto completo: 1 Base de dados: MEDLINE Assunto principal: Peptídeos beta-Amiloides / Potenciação de Longa Duração / Doença de Alzheimer / Proteínas Priônicas / Plasticidade Neuronal Limite: Animals / Humans Idioma: En Ano de publicação: 2016 Tipo de documento: Article