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Impaired Endothelial Nitric Oxide Synthase Homodimer Formation Triggers Development of Transplant Vasculopathy - Insights from a Murine Aortic Transplantation Model.
Oberhuber, Rupert; Riede, Gregor; Cardini, Benno; Bernhard, David; Messner, Barbara; Watschinger, Katrin; Steger, Christina; Brandacher, Gerald; Pratschke, Johann; Golderer, Georg; Werner, Ernst R; Maglione, Manuel.
Afiliação
  • Oberhuber R; Centre of Operative Medicine, Department of Visceral, Transplant and Thoracic Surgery, Medical University Innsbruck, Innsbruck, Austria.
  • Riede G; Centre of Operative Medicine, Department of Visceral, Transplant and Thoracic Surgery, Medical University Innsbruck, Innsbruck, Austria.
  • Cardini B; Centre of Operative Medicine, Department of Visceral, Transplant and Thoracic Surgery, Medical University Innsbruck, Innsbruck, Austria.
  • Bernhard D; Cardiac Surgery Research Laboratory, University Clinic for Cardiac Surgery, Medical University Innsbruck, Innsbruck, Austria.
  • Messner B; Cardiac Surgery Research Laboratory, Department of Surgery, Vienna Medical University, Austria.
  • Watschinger K; Division of Biological Chemistry, Biocenter, Medical University Innsbruck, Innsbruck, Austria.
  • Steger C; Institute of Pathology, Academic Teaching Hospital Feldkirch, Feldkirch, Austria.
  • Brandacher G; Centre of Operative Medicine, Department of Visceral, Transplant and Thoracic Surgery, Medical University Innsbruck, Innsbruck, Austria.
  • Pratschke J; Department of Plastic and Reconstructive Surgery, Vascularized Composite Allotransplantation (VCA) Laboratory, Johns Hopkins University School of Medicine, Baltimore, Maryland, USA.
  • Golderer G; Centre of Operative Medicine, Department of Visceral, Transplant and Thoracic Surgery, Medical University Innsbruck, Innsbruck, Austria.
  • Werner ER; Department of General-, Visceral- and Transplantation Surgery, Charité, Campus Virchow Klinikum, Berlin, Germany.
  • Maglione M; Division of Biological Chemistry, Biocenter, Medical University Innsbruck, Innsbruck, Austria.
Sci Rep ; 6: 37917, 2016 11 24.
Article em En | MEDLINE | ID: mdl-27883078
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Texto completo: 1 Base de dados: MEDLINE Assunto principal: Aorta / Traumatismo por Reperfusão / Óxido Nítrico Sintase Tipo III Tipo de estudo: Prognostic_studies Limite: Animals Idioma: En Ano de publicação: 2016 Tipo de documento: Article
Texto completo
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Texto completo: 1 Base de dados: MEDLINE Assunto principal: Aorta / Traumatismo por Reperfusão / Óxido Nítrico Sintase Tipo III Tipo de estudo: Prognostic_studies Limite: Animals Idioma: En Ano de publicação: 2016 Tipo de documento: Article