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Endothelial specific SIRT3 deletion impairs glycolysis and angiogenesis and causes diastolic dysfunction.
He, Xiaochen; Zeng, Heng; Chen, Sean T; Roman, Richard J; Aschner, Judy L; Didion, Sean; Chen, Jian-Xiong.
Afiliação
  • He X; Department of Pharmacology and Toxicology, University of Mississippi Medical Center, Jackson, MS, USA.
  • Zeng H; Department of Pharmacology and Toxicology, University of Mississippi Medical Center, Jackson, MS, USA.
  • Chen ST; Duke University School of Medicine, Durham, NC, USA.
  • Roman RJ; Department of Pharmacology and Toxicology, University of Mississippi Medical Center, Jackson, MS, USA.
  • Aschner JL; Department of Pediatrics, Albert Einstein College of Medicine and the Children's Hospital at Montefiore, Bronx, NY, USA.
  • Didion S; Department of Pharmacology and Toxicology, University of Mississippi Medical Center, Jackson, MS, USA.
  • Chen JX; Department of Pharmacology and Toxicology, University of Mississippi Medical Center, Jackson, MS, USA. Electronic address: JChen3@umc.edu.
J Mol Cell Cardiol ; 112: 104-113, 2017 11.
Article em En | MEDLINE | ID: mdl-28935506
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Texto completo: 1 Base de dados: MEDLINE Assunto principal: Deleção de Genes / Neovascularização Fisiológica / Diástole / Sirtuína 3 / Glicólise / Coração Tipo de estudo: Etiology_studies Limite: Animals Idioma: En Ano de publicação: 2017 Tipo de documento: Article
Texto completo
Imprimir
XML
PubMed Links
Buscar no Google

Texto completo: 1 Base de dados: MEDLINE Assunto principal: Deleção de Genes / Neovascularização Fisiológica / Diástole / Sirtuína 3 / Glicólise / Coração Tipo de estudo: Etiology_studies Limite: Animals Idioma: En Ano de publicação: 2017 Tipo de documento: Article