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NLRC3 Restrains Responses to a T.
Holley, Caroline L; Coll, Rebecca C; Schroder, Kate.
Afiliação
  • Holley CL; Institute for Molecular Bioscience and IMB Centre for Inflammation and Disease Research, University of Queensland, St. Lucia, QLD, Australia.
  • Coll RC; Institute for Molecular Bioscience and IMB Centre for Inflammation and Disease Research, University of Queensland, St. Lucia, QLD, Australia.
  • Schroder K; Institute for Molecular Bioscience and IMB Centre for Inflammation and Disease Research, University of Queensland, St. Lucia, QLD, Australia. Electronic address: k.schroder@imb.uq.edu.au.
Immunity ; 49(6): 989-991, 2018 12 18.
Article em En | MEDLINE | ID: mdl-30566886
ABSTRACT
The NOD-like receptor protein NLRC3 attenuates myeloid cell inflammatory responses. In this issue of Immunity, Uchimura et al. (2018) reveal additional T-cell-intrinsic functions for NLRC3 in restricting T cell metabolism, T helper 1 and T helper 17 cell responses, and antiviral and autoimmune responses.
Assuntos

Texto completo: 1 Base de dados: MEDLINE Assunto principal: Autoimunidade / Peptídeos e Proteínas de Sinalização Intercelular Idioma: En Ano de publicação: 2018 Tipo de documento: Article

Texto completo: 1 Base de dados: MEDLINE Assunto principal: Autoimunidade / Peptídeos e Proteínas de Sinalização Intercelular Idioma: En Ano de publicação: 2018 Tipo de documento: Article