Cancer-bone microenvironmental interactions promotes STAT3 signaling.

Henderson, Veronica M; Hawsawi, Ohuod; Burton, Liza J; Campbell, Taaliah; Trice, Kennedi; Dougan, Jodi; Howard, Simone M; Odero-Marah, Valerie A

Henderson, Veronica M; Hawsawi, Ohuod; Burton, Liza J; Campbell, Taaliah; Trice, Kennedi; Dougan, Jodi; Howard, Simone M; Odero-Marah, Valerie A.

Afiliação

Henderson VM; Department of Biological Sciences, Center for Cancer Research and Therapeutic Development, Clark Atlanta University, Atlanta, Georgia.
Hawsawi O; Department of Biological Sciences, Center for Cancer Research and Therapeutic Development, Clark Atlanta University, Atlanta, Georgia.
Burton LJ; Department of Biological Sciences, Center for Cancer Research and Therapeutic Development, Clark Atlanta University, Atlanta, Georgia.
Campbell T; Department of Biological Sciences, Center for Cancer Research and Therapeutic Development, Clark Atlanta University, Atlanta, Georgia.
Trice K; Department of Biological Sciences, Center for Cancer Research and Therapeutic Development, Clark Atlanta University, Atlanta, Georgia.
Dougan J; Department of Biological Sciences, Center for Cancer Research and Therapeutic Development, Clark Atlanta University, Atlanta, Georgia.
Howard SM; Department of Biological Sciences, Center for Cancer Research and Therapeutic Development, Clark Atlanta University, Atlanta, Georgia.
Odero-Marah VA; Department of Biological Sciences, Center for Cancer Research and Therapeutic Development, Clark Atlanta University, Atlanta, Georgia.

Mol Carcinog ; 58(8): 1349-1361, 2019 08.

Article em En | MEDLINE | ID: mdl-31045290

Assuntos

Neoplasias Ósseas/patologia; Neoplasias Ósseas/secundário; Neoplasias da Próstata/patologia; Fator de Transcrição STAT3/metabolismo; Microambiente Tumoral/fisiologia; Animais; Osso e Ossos/patologia; Cálcio/metabolismo; Catepsina L/antagonistas & inibidores; Linhagem Celular Tumoral; Movimento Celular/fisiologia; Proliferação de Células/fisiologia; Durapatita/farmacologia; Ácido Egtázico/farmacologia; Células HEK293; Humanos; Masculino; Camundongos; Camundongos Endogâmicos BALB C; Camundongos Nus; Fosforilação; Piridinas; Interferência de RNA; RNA Interferente Pequeno/genética; Fator de Transcrição STAT3/antagonistas & inibidores; Transdução de Sinais; Fatores de Transcrição da Família Snail/genética; Tirfostinas

Palavras-chave

Snail; cancer-bone microenvironmental interactions; cell migration; epithelial to mesenchymal transition; hydroxyapatite; prostate cancer cells; signal transducer and activator of transcription 3

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Texto completo: 1 Base de dados: MEDLINE Assunto principal: Neoplasias da Próstata / Neoplasias Ósseas / Fator de Transcrição STAT3 / Microambiente Tumoral Limite: Animals / Humans / Male Idioma: En Ano de publicação: 2019 Tipo de documento: Article

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