Metabolic, Phenotypic, and Neuropathological Characterization of the Tg4-42 Mouse Model for Alzheimer's Disease.

Hinteregger, Barbara; Loeffler, Tina; Flunkert, Stefanie; Neddens, Joerg; Bayer, Thomas A; Madl, Tobias; Hutter-Paier, Birgit

Hinteregger, Barbara; Loeffler, Tina; Flunkert, Stefanie; Neddens, Joerg; Bayer, Thomas A; Madl, Tobias; Hutter-Paier, Birgit.

Afiliação

Hinteregger B; QPS Austria GmbH, Grambach, Austria.
Loeffler T; Gottfried Schatz Research Center (for Cell Signaling, Metabolism and Aging), Division of Molecular Biology and Biochemistry, Medical University of Graz, Graz, Austria.
Flunkert S; QPS Austria GmbH, Grambach, Austria.
Neddens J; QPS Austria GmbH, Grambach, Austria.
Bayer TA; QPS Austria GmbH, Grambach, Austria.
Madl T; Department of Psychiatry and Psychotherapy, Division of Molecular Psychiatry, University Medical Center, Göttingen (UMG), Göttingen, Germany.
Hutter-Paier B; Gottfried Schatz Research Center (for Cell Signaling, Metabolism and Aging), Division of Molecular Biology and Biochemistry, Medical University of Graz, Graz, Austria.

J Alzheimers Dis ; 80(3): 1151-1168, 2021.

Article em En | MEDLINE | ID: mdl-33646155

Assuntos

Doença de Alzheimer/metabolismo; Doença de Alzheimer/patologia; Peptídeos beta-Amiloides/genética; Modelos Animais de Doenças; Animais; Humanos; Masculino; Camundongos; Camundongos Endogâmicos C57BL; Camundongos Transgênicos; Fenótipo

Palavras-chave

Alzheimer's disease; behavior; biomarkers; neuroinflammation; neuronal degeneration; nuclear magnetic resonance

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Texto completo: 1 Base de dados: MEDLINE Assunto principal: Peptídeos beta-Amiloides / Modelos Animais de Doenças / Doença de Alzheimer Limite: Animals / Humans / Male Idioma: En Ano de publicação: 2021 Tipo de documento: Article

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