Autoimmune neuroinflammation triggers mitochondrial oxidation in oligodendrocytes.

Steudler, Jasmin; Ecott, Timothy; Ivan, Daniela C; Bouillet, Elisa; Walthert, Sabrina; Berve, Kristina; Dick, Tobias P; Engelhardt, Britta; Locatelli, Giuseppe

Steudler, Jasmin; Ecott, Timothy; Ivan, Daniela C; Bouillet, Elisa; Walthert, Sabrina; Berve, Kristina; Dick, Tobias P; Engelhardt, Britta; Locatelli, Giuseppe.

Afiliação

Steudler J; Theodor Kocher Institute, University of Bern, Bern, Switzerland.
Ecott T; Theodor Kocher Institute, University of Bern, Bern, Switzerland.
Ivan DC; Theodor Kocher Institute, University of Bern, Bern, Switzerland.
Bouillet E; Theodor Kocher Institute, University of Bern, Bern, Switzerland.
Walthert S; Theodor Kocher Institute, University of Bern, Bern, Switzerland.
Berve K; Theodor Kocher Institute, University of Bern, Bern, Switzerland.
Dick TP; Division of Redox Regulation, DKFZ-ZMBH Alliance, German Cancer Research Center (DKFZ), Heidelberg, Germany.
Engelhardt B; Theodor Kocher Institute, University of Bern, Bern, Switzerland.
Locatelli G; Theodor Kocher Institute, University of Bern, Bern, Switzerland.

Glia ; 70(11): 2045-2061, 2022 11.

Article em En | MEDLINE | ID: mdl-35762739

Assuntos

Encefalomielite Autoimune Experimental; Esclerose Múltipla; Animais; Encefalomielite Autoimune Experimental/patologia; Camundongos; Camundongos Endogâmicos C57BL; Mitocôndrias/metabolismo; Esclerose Múltipla/patologia; Doenças Neuroinflamatórias; Oligodendroglia/metabolismo; Oxirredução; Medula Espinal/metabolismo

Palavras-chave

experimental autoimmune encephalomyelitis; mitochondria; multiple sclerosis; myelin; neurodegeneration; oligodendrocyte; reactive species

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Texto completo: 1 Base de dados: MEDLINE Assunto principal: Encefalomielite Autoimune Experimental / Esclerose Múltipla Limite: Animals Idioma: En Ano de publicação: 2022 Tipo de documento: Article

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