摘要
@#ObjectiveTo explore the influence of breath muscles functional training on convalescent patients with chronic obstructive pulmonary disease(COPD).Methods56 COPD patients had breath muscles functional training for 1 year and clinical indexes,including cold time,time in hospital,hemoglobin,blood gas analysis and hemorheology were observed.ResultsAfter breath muscles functional training,the cold time and time in hospital had shortened;hemoglobin,blood gas analysis and hemorheology of patients had ameliorated obviously(P<0.01).ConclusionBreath muscles functional training has affirmatively effect of boosting up corporeity,ameliorating lack of oxygen,adjusting internal environment on convalescent patients with COPD.
摘要
Neuropathies are complications of cardiac surgery. The reported frequency of nerve injuries varies from 1. 5% to 2. 4% for the brachial plexus and 10% to 60% for the phrenic nerve. But the recurrent laryngea! nerve,lower limb (saphenous) nerves, and the sympathetic chain are less frequently injured. The reasons of neuropathy are mainly positioning and topical hypothermia for myocardial protection. The overall prognosis of neuropathies after cardiac surgery is good, and they need no special treatment. However, prolonged recovery of phrenic neuropathy did occurr.
摘要
AIM: To study the dynamic changes of amino acid neurotransmitters during cerebral ischemia and reperfusion in awake rats. METHODS: Model of cerebral ischemia and reperfusion in awake rats was replicated. Glutamate (Glu), aspartate (Asp), ?-aminobutyric acid (GABA), glycine (Gly), taurine (Tau), alanine (Ala), serine (Ser), threonine (Thr) and glutamine (Gln) concentrations were measured with microdialysis, and excitotoxic index (EI) was calculated in dialysates of hippocampus, neo-cortex and striatum. RESULTS: The significant increases in extracellular not only excitatory amino acid neurotransmitters-Glu and Asp, and their neuromodulator-Gly, but also inhibitory amino acid neurotransmitter-GABA and its neuromodulator-Tau and Ala were observed. However, the EI, representing the balance of excitatory and inhibitory neurotransmitters, was significantly increased during ischemia. CONCLUSION: The results suggest that the elevated Glu and Asp levels during ischemia are insufficient to independently engender ischemic damage, and other neurotransmitters or other factors may play an important role in modulating the excitotoxic effects of Glu and Asp.