Calcium/Calmodulin Mediates Cardiomyocyte Ferroptosis in Myocardial Infarction Through Iron Overload / 中国生物化学与分子生物学报
Chinese Journal of Biochemistry and Molecular Biology
; (12): 1023-1035, 2023.
Article
ي Zh
| WPRIM
| ID: wpr-1015622
المكتبة المسؤولة:
WPRO
ABSTRACT
Myocardial infarction(MI), an acute coronary syndrome that poses a serious risk to human health, involves multiple pathophysiological processes, including calcium overload. Existing therapeutic approaches and preventive measures have limitations and cannot effectively repair myocardial cells with poor regenerative potential. Exploring multiple programmed modes of cardiomyocyte death could help find potential targets for the treatment of myocardial infarction, and the potential role of ferroptosis as a novel mode of cell death in myocardial infarction has attracted great attention. The aim of this study was to investigate whether Ca
النص الكامل:
1
الفهرس:
WPRIM
اللغة:
Zh
مجلة:
Chinese Journal of Biochemistry and Molecular Biology
السنة:
2023
نوع:
Article