A Case of Cerebellar Dysfunction After Acute Organotin Poisoning / 대한산업의학회지
Korean Journal of Occupational and Environmental Medicine
; : 289-292, 2009.
Article
ي Ko
| WPRIM
| ID: wpr-177606
المكتبة المسؤولة:
WPRO
ABSTRACT
BACKGROUND:
We performed a follow-up study of an acute organotin poisoning case on the neurological sequelae that remained after 3 years since diagnosis. CASE In the previous study, a 43-year-old male was reported for acute organotin poisoning. After 3 years, the neurological sequelae were studied using neurological examination, cognitive tests, neurobehavioral tests (by CAYSYS 2000(TM)), brain magnetic resonance imaging (MRI), and 18F-fluorodeoxyglucose (18F-FDG) PET/CT. Upon neurological examination and other tests, orientation was improved compared to that 3 years before, but cerebellar ataxia, dysmetria and dysdiadochokinesia remained the same, with little change. Brain MRI showed cerebellar atrophy. 18F-FDG PET/CT revealed mildly decreased metabolic activity in the pons and in both cerebellar hemispheres.DISCUSSION:
This is the first brain MRI study on cerebellar atrophy caused by organotin poisoning. Cerebellar ataxia was confirmed by decreased metabolic activity in 18F-FDG PET/CT.Key words
النص الكامل:
1
الفهرس:
WPRIM
الموضوع الرئيسي:
Orientation
/
Atrophy
/
Brain
/
Pons
/
Magnetic Resonance Imaging
/
Cerebellar Ataxia
/
Cerebellar Diseases
/
Follow-Up Studies
/
Fluorodeoxyglucose F18
/
Neurologic Examination
نوع الدراسة:
Observational_studies
/
Prognostic_studies
المحددات:
Adult
/
Humans
/
Male
اللغة:
Ko
مجلة:
Korean Journal of Occupational and Environmental Medicine
السنة:
2009
نوع:
Article