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Myasthenia in Acquired Neuromyotonia
Article 在 En | WPRIM | ID: wpr-113288
Responsible library: WPRO
ABSTRACT
BACKGROUND: Acquired neuromyotonia (NMT) forms part of the spectrum of acquired peripheral nerve hyperexcitability syndrome, and is thought to be caused by antibodies to voltage-gated potassium channels (VGKC). Exertional weakness is unusual unless autoimmune myasthenia gravis (MG) is superimposed. CASE REPORT: A case of acquired NMT accompanied by exertional weakness without coexistence of seropositive MG is reported herein. CONCLUSIONS: Clinical and electrophysiological observations suggest that the cholinergic overactivity in NMT can compromise the safety factor sufficiently to cause a defect in neuromuscular junction transmission.
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全文: 1 索引: WPRIM 主要主题: Peripheral Nerves / Isaacs Syndrome / Potassium Channels, Voltage-Gated / Antibodies / Myasthenia Gravis / Neuromuscular Junction 语言: En 期刊: Journal of Clinical Neurology 年: 2014 类型: Article
全文: 1 索引: WPRIM 主要主题: Peripheral Nerves / Isaacs Syndrome / Potassium Channels, Voltage-Gated / Antibodies / Myasthenia Gravis / Neuromuscular Junction 语言: En 期刊: Journal of Clinical Neurology 年: 2014 类型: Article