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1.
Expression of murine muscle-enriched A-type lamin-interacting protein (MLIP) is regulated by tissue-specific alternative transcription start sites.
J Biol Chem
; 293(51): 19761-19770, 2018 12 21.
Artículo
en Inglés
| MEDLINE | ID: mdl-30389785
2.
In Vitro Modeling of Congenital Heart Defects Associated with an NKX2-5 Mutation Revealed a Dysregulation in BMP/Notch-Mediated Signaling.
Stem Cells
; 36(4): 514-526, 2018 04.
Artículo
en Inglés
| MEDLINE | ID: mdl-29282804
3.
Deletion of MLIP (muscle-enriched A-type lamin-interacting protein) leads to cardiac hyperactivation of Akt/mammalian target of rapamycin (mTOR) and impaired cardiac adaptation.
J Biol Chem
; 290(44): 26699-714, 2015 Oct 30.
Artículo
en Inglés
| MEDLINE | ID: mdl-26359501
4.
Heterozygous LmnadelK32 mice develop dilated cardiomyopathy through a combined pathomechanism of haploinsufficiency and peptide toxicity.
Hum Mol Genet
; 22(15): 3152-64, 2013 Aug 01.
Artículo
en Inglés
| MEDLINE | ID: mdl-23575224
5.
DelK32-lamin A/C has abnormal location and induces incomplete tissue maturation and severe metabolic defects leading to premature death.
Hum Mol Genet
; 21(5): 1037-48, 2012 Mar 01.
Artículo
en Inglés
| MEDLINE | ID: mdl-22090424
6.
'State-of-the-heart' of cardiac laminopathies.
Curr Opin Cardiol
; 28(3): 297-304, 2013 May.
Artículo
en Inglés
| MEDLINE | ID: mdl-23455585
7.
Filling the drug discovery gap: is high-content screening the missing link?
Curr Opin Pharmacol
; 42: 40-45, 2018 10.
Artículo
en Inglés
| MEDLINE | ID: mdl-30032033
8.
Mutation in lamin A/C sensitizes the myocardium to exercise-induced mechanical stress but has no effect on skeletal muscles in mouse.
Neuromuscul Disord
; 26(8): 490-9, 2016 08.
Artículo
en Inglés
| MEDLINE | ID: mdl-27287550
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