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1.
Non-motor symptoms associated with progressive loss of dopaminergic neurons in a mouse model of Parkinson's disease.
Front Neurosci
; 18: 1375265, 2024.
Artículo
en Inglés
| MEDLINE | ID: mdl-38745938
2.
Genetic lesions of the noradrenergic system trigger induction of oxidative stress and inflammation in the ventral midbrain.
Neurochem Int
; 155: 105302, 2022 05.
Artículo
en Inglés
| MEDLINE | ID: mdl-35150790
3.
The influence of CaMKII and ERK phosphorylation on BDNF changes observed in mice selectively devoid of CREB in serotonergic or noradrenergic neurons.
Pharmacol Rep
; 71(5): 753-761, 2019 Oct.
Artículo
en Inglés
| MEDLINE | ID: mdl-31351316
4.
Stimulation of noradrenergic transmission by reboxetine is beneficial for a mouse model of progressive parkinsonism.
Sci Rep
; 9(1): 5262, 2019 03 27.
Artículo
en Inglés
| MEDLINE | ID: mdl-30918302
5.
Selective Depletion of CREB in Serotonergic Neurons Affects the Upregulation of Brain-Derived Neurotrophic Factor Evoked by Chronic Fluoxetine Treatment.
Front Neurosci
; 12: 637, 2018.
Artículo
en Inglés
| MEDLINE | ID: mdl-30294251
6.
Lack of riluzole efficacy in the progression of the neurodegenerative phenotype in a new conditional mouse model of striatal degeneration.
PeerJ
; 5: e3240, 2017.
Artículo
en Inglés
| MEDLINE | ID: mdl-28462043
7.
Transgenic mice lacking CREB and CREM in noradrenergic and serotonergic neurons respond differently to common antidepressants on tail suspension test.
Sci Rep
; 7(1): 13515, 2017 10 18.
Artículo
en Inglés
| MEDLINE | ID: mdl-29044198
8.
Disruption of glucocorticoid receptors in the noradrenergic system leads to BDNF up-regulation and altered serotonergic transmission associated with a depressive-like phenotype in female GR(DBHCre) mice.
Pharmacol Biochem Behav
; 137: 69-77, 2015 Oct.
Artículo
en Inglés
| MEDLINE | ID: mdl-26261018
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