Búsqueda | Biblioteca Virtual en Salud Fronteriza

1.

Aberrant epithelial polarity cues drive the development of precancerous airway lesions.

Tilston-Lunel, Andrew; Mazzilli, Sarah; Kingston, Nathan M; Szymaniak, Aleksander D; Hicks-Berthet, Julia; Kern, Joseph G; Abo, Kristine; Reid, Mary E; Perdomo, Catalina; Wilson, Andrew A; Spira, Avrum; Beane, Jennifer; Varelas, Xaralabos.

Proc Natl Acad Sci U S A ; 118(18)2021 05 04.

Artículo en Inglés | MEDLINE | ID: mdl-33903236

2.

Inactivation of LATS1/2 drives luminal-basal plasticity to initiate basal-like mammary carcinomas.

Kern, Joseph G; Tilston-Lunel, Andrew M; Federico, Anthony; Ning, Boting; Mueller, Amy; Peppler, Grace B; Stampouloglou, Eleni; Cheng, Nan; Johnson, Randy L; Lenburg, Marc E; Beane, Jennifer E; Monti, Stefano; Varelas, Xaralabos.

Nat Commun ; 13(1): 7198, 2022 11 28.

Artículo en Inglés | MEDLINE | ID: mdl-36443313

3.

The Tumor Suppressor CYLD Inhibits Mammary Epithelial to Mesenchymal Transition by the Coordinated Inhibition of YAP/TAZ and TGFï¢ Signaling.

Pseftogas, Athanasios; Xanthopoulos, Konstantinos; Poutahidis, Theofilos; Ainali, Chrysanthi; Dafou, Dimitra; Panteris, Emmanuel; Kern, Joseph G; Varelas, Xaralabos; Hardas, Alexander; Gonidas, Christos; Tsingotjidou, Anastasia; Hatzivassiliou, Eudoxia; Mosialos, George.

Cancers (Basel) ; 12(8)2020 Jul 24.

Artículo en Inglés | MEDLINE | ID: mdl-32722292

4.

TAZ Forces Lateral Inhibition.

Kern, Joseph G; Varelas, Xaralabos.

Dev Cell ; 48(6): 748-750, 2019 03 25.

Artículo en Inglés | MEDLINE | ID: mdl-30913406

5.

CaDrA: A Computational Framework for Performing Candidate Driver Analyses Using Genomic Features.

Kartha, Vinay K; Sebastiani, Paola; Kern, Joseph G; Zhang, Liye; Varelas, Xaralabos; Monti, Stefano.

Front Genet ; 10: 121, 2019.

Artículo en Inglés | MEDLINE | ID: mdl-30838036

6.

The emerging role and targetability of the TCA cycle in cancer metabolism.

Anderson, Nicole M; Mucka, Patrick; Kern, Joseph G; Feng, Hui.

Protein Cell ; 9(2): 216-237, 2018 02.

Artículo en Inglés | MEDLINE | ID: mdl-28748451

7.

Transcriptional-mediated effects of radiation on the expression of immune susceptibility markers in melanoma.

Werner, Lauryn R; Kler, Jasdeep S; Gressett, Monica M; Riegert, Maureen; Werner, Lindsey K; Heinze, Clinton M; Kern, Joseph G; Abbariki, Mahyar; Erbe, Amy K; Patel, Ravi B; Sriramaneni, Raghava N; Harari, Paul M; Morris, Zachary S.

Radiother Oncol ; 124(3): 418-426, 2017 09.

Artículo en Inglés | MEDLINE | ID: mdl-28893414

Biblioteca Virtual en Salud Fronteriza

México - Estados Unidos

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Aberrant epithelial polarity cues drive the development of precancerous airway lesions.

Inactivation of LATS1/2 drives luminal-basal plasticity to initiate basal-like mammary carcinomas.

The Tumor Suppressor CYLD Inhibits Mammary Epithelial to Mesenchymal Transition by the Coordinated Inhibition of YAP/TAZ and TGFï¢ Signaling.

TAZ Forces Lateral Inhibition.

CaDrA: A Computational Framework for Performing Candidate Driver Analyses Using Genomic Features.

The emerging role and targetability of the TCA cycle in cancer metabolism.

Transcriptional-mediated effects of radiation on the expression of immune susceptibility markers in melanoma.

Biblioteca Virtual en Salud Fronteriza

México - Estados Unidos

Su selección (0)

Formato de exportación:

Exportar:

Aberrant epithelial polarity cues drive the development of precancerous airway lesions.

Inactivation of LATS1/2 drives luminal-basal plasticity to initiate basal-like mammary carcinomas.

The Tumor Suppressor CYLD Inhibits Mammary Epithelial to Mesenchymal Transition by the Coordinated Inhibition of YAP/TAZ and TGFï¢ Signaling.

TAZ Forces Lateral Inhibition.

CaDrA: A Computational Framework for Performing Candidate Driver Analyses Using Genomic Features.

The emerging role and targetability of the TCA cycle in cancer metabolism.

Transcriptional-mediated effects of radiation on the expression of immune susceptibility markers in melanoma.

The Tumor Suppressor CYLD Inhibits Mammary Epithelial to Mesenchymal Transition by the Coordinated Inhibition of YAP/TAZ and TGFï¢ Signaling.