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1.
T-cell exhaustion induced by continuous bispecific molecule exposure is ameliorated by treatment-free intervals.
Blood
; 140(10): 1104-1118, 2022 09 08.
Artículo
en Inglés
| MEDLINE | ID: mdl-35878001
2.
CD33 BiTE® molecule-mediated immune synapse formation and subsequent T-cell activation is determined by the expression profile of activating and inhibitory checkpoint molecules on AML cells.
Cancer Immunol Immunother
; 72(7): 2499-2512, 2023 Jul.
Artículo
en Inglés
| MEDLINE | ID: mdl-37041225
3.
WT1 and DNMT3A play essential roles in the growth of certain patient AML cells in mice.
Blood
; 141(8): 955-960, 2023 02 23.
Artículo
en Inglés
| MEDLINE | ID: mdl-36256915
4.
Bifunctional PD-1 × αCD3 × αCD33 fusion protein reverses adaptive immune escape in acute myeloid leukemia.
Blood
; 132(23): 2484-2494, 2018 12 06.
Artículo
en Inglés
| MEDLINE | ID: mdl-30275109
5.
Correction to: CD33 BiTE® molecule-mediated immune synapse formation and subsequent T-cell activation is determined by the expression profile of activating and inhibitory checkpoint molecules on AML cells.
Cancer Immunol Immunother
; 72(7): 2513-2514, 2023 Jul.
Artículo
en Inglés
| MEDLINE | ID: mdl-37154851
6.
SIRPα-αCD123 fusion antibodies targeting CD123 in conjunction with CD47 blockade enhance the clearance of AML-initiating cells.
J Hematol Oncol
; 14(1): 155, 2021 09 27.
Artículo
en Inglés
| MEDLINE | ID: mdl-34579739
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