Tumor necrosis factor-induced cytotoxicity is not related to rates of mitochondrial morphological abnormalities or autophagy-changes that can be mediated by TNFR-I or TNFR-II.

Prins, J B; Ledgerwood, E C; Ameloot, P; Vandenabeele, P; Faraco, P R; Bright, N A; O'Rahilly, S; Bradley, J R

Prins, J B; Ledgerwood, E C; Ameloot, P; Vandenabeele, P; Faraco, P R; Bright, N A; O'Rahilly, S; Bradley, J R.

Affiliation

Prins JB; Department of Medicine, University of Cambridge, UK.

Biosci Rep ; 18(6): 329-40, 1998 Dec.

Article in En | MEDLINE | ID: mdl-10357175

Subject(s)

Antigens, CD/metabolism; Autophagy/physiology; Mitochondria/drug effects; Receptors, Tumor Necrosis Factor/metabolism; Tumor Necrosis Factor-alpha/pharmacology; 3T3 Cells/drug effects; 3T3 Cells/metabolism; 3T3 Cells/ultrastructure; Animals; Apoptosis/drug effects; Autophagy/drug effects; Cell Line; Humans; Mice; Mitochondria/ultrastructure; Receptors, Tumor Necrosis Factor, Type I; Receptors, Tumor Necrosis Factor, Type II; Tumor Necrosis Factor-alpha/physiology

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Database: MEDLINE Main subject: Autophagy / Antigens, CD / Tumor Necrosis Factor-alpha / Receptors, Tumor Necrosis Factor / Mitochondria Limits: Animals / Humans Language: En Year: 1998 Type: Article

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