Loss of orphan receptor germ cell nuclear factor function results in ectopic development of the tail bud and a novel posterior truncation.

Chung, A C; Katz, D; Pereira, F A; Jackson, K J; DeMayo, F J; Cooney, A J; O'Malley, B W

Chung, A C; Katz, D; Pereira, F A; Jackson, K J; DeMayo, F J; Cooney, A J; O'Malley, B W.

Affiliation

Chung AC; Department of Molecular and Cellular Biology, Baylor College of Medicine, Houston, Texas 77030, USA.

Mol Cell Biol ; 21(2): 663-77, 2001 Jan.

Article in En | MEDLINE | ID: mdl-11134352

Subject(s)

Choristoma/embryology; DNA-Binding Proteins/metabolism; Gene Deletion; Limb Buds/abnormalities; Receptors, Cytoplasmic and Nuclear/metabolism; Tail/abnormalities; Animals; Cell Differentiation; Choristoma/metabolism; DNA-Binding Proteins/genetics; Embryonic and Fetal Development; Fetal Death; Gene Expression Regulation, Developmental; Gene Targeting; Genetic Markers; Histocytochemistry; In Situ Hybridization; Limb Buds/cytology; Limb Buds/embryology; Limb Buds/metabolism; Mice; Mice, Knockout; Nuclear Receptor Subfamily 6, Group A, Member 1; RNA, Messenger/analysis; RNA, Messenger/genetics; Receptors, Cytoplasmic and Nuclear/genetics; Recombination, Genetic/genetics; Somites/cytology; Somites/metabolism; Tail/cytology; Tail/embryology; Tail/metabolism

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Full text: 1 Database: MEDLINE Main subject: Tail / Choristoma / Gene Deletion / Receptors, Cytoplasmic and Nuclear / Limb Buds / DNA-Binding Proteins Type of study: Prognostic_studies Limits: Animals Language: En Year: 2001 Type: Article

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