Hyperosmotic stress activates p65/RelB NFkappaB in cultured cardiomyocytes with dichotomic actions on caspase activation and cell death.

Eisner, Verónica; Quiroga, Clara; Criollo, Alfredo; Eltit, José Miguel; Chiong, Mario; Parra, Valentina; Hidalgo, Karla; Toro, Barbra; Díaz-Araya, Guillermo; Lavandero, Sergio

Eisner, Verónica; Quiroga, Clara; Criollo, Alfredo; Eltit, José Miguel; Chiong, Mario; Parra, Valentina; Hidalgo, Karla; Toro, Barbra; Díaz-Araya, Guillermo; Lavandero, Sergio.

Affiliation

Eisner V; Faculty of Chemical and Pharmaceutical Sciences, University of Chile, Santiago.

FEBS Lett ; 580(14): 3469-76, 2006 Jun 12.

Article in En | MEDLINE | ID: mdl-16716309

Subject(s)

Apoptosis; Caspases/metabolism; Myocardium/metabolism; Transcription Factor RelB/metabolism; Animals; Cells, Cultured; Enzyme Activation; Myocardium/cytology; Myocardium/enzymology; Osmotic Pressure; Rats

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Database: MEDLINE Main subject: Apoptosis / Caspases / Transcription Factor RelB / Myocardium Type of study: Prognostic_studies Limits: Animals Language: En Year: 2006 Type: Article

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