Triadins modulate intracellular Ca(2+) homeostasis but are not essential for excitation-contraction coupling in skeletal muscle.

Shen, Xiaohua; Franzini-Armstrong, Clara; Lopez, Jose R; Jones, Larry R; Kobayashi, Yvonne M; Wang, Ying; Kerrick, W Glenn L; Caswell, Anthony H; Potter, James D; Miller, Todd; Allen, Paul D; Perez, Claudio F

Shen, Xiaohua; Franzini-Armstrong, Clara; Lopez, Jose R; Jones, Larry R; Kobayashi, Yvonne M; Wang, Ying; Kerrick, W Glenn L; Caswell, Anthony H; Potter, James D; Miller, Todd; Allen, Paul D; Perez, Claudio F.

Affiliation

Shen X; Department of Anesthesiology, Brigham and Women's Hospital, Boston, MA 02115, USA.

J Biol Chem ; 282(52): 37864-74, 2007 Dec 28.

Article in En | MEDLINE | ID: mdl-17981799

Subject(s)

Calcium/metabolism; Calsequestrin/genetics; Carrier Proteins/genetics; Carrier Proteins/physiology; Gene Expression Regulation; Muscle Proteins/genetics; Muscle Proteins/physiology; Muscle, Skeletal/metabolism; Animals; Calcium-Binding Proteins/genetics; Exons; Membrane Proteins/genetics; Mice; Mice, Knockout; Mice, Transgenic; Mixed Function Oxygenases/genetics; Models, Biological; Muscle Fibers, Skeletal/metabolism; Potassium Chloride/metabolism; Sarcoplasmic Reticulum/metabolism

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Database: MEDLINE Main subject: Calsequestrin / Carrier Proteins / Gene Expression Regulation / Calcium / Muscle, Skeletal / Muscle Proteins Type of study: Prognostic_studies Limits: Animals Language: En Year: 2007 Type: Article

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