Acute atrial tachyarrhythmia induces angiotensin II type 1 receptor-mediated oxidative stress and microvascular flow abnormalities in the ventricles.

Goette, Andreas; Bukowska, Alicja; Dobrev, Dobromir; Pfeiffenberger, Jan; Morawietz, Henning; Strugala, Denis; Wiswedel, Ingrid; Röhl, Friedrich-Wilhelm; Wolke, Carmen; Bergmann, Sybille; Bramlage, Peter; Ravens, Ursula; Lendeckel, Uwe

Goette, Andreas; Bukowska, Alicja; Dobrev, Dobromir; Pfeiffenberger, Jan; Morawietz, Henning; Strugala, Denis; Wiswedel, Ingrid; Röhl, Friedrich-Wilhelm; Wolke, Carmen; Bergmann, Sybille; Bramlage, Peter; Ravens, Ursula; Lendeckel, Uwe.

Affiliation

Goette A; Division of Cardiology, University Hospital Magdeburg, Otto-von-Guericke University, Leipzigerstr. 44, 39120 Magdeburg, Germany. andreas.goette@med.ovgu.de

Eur Heart J ; 30(11): 1411-20, 2009 Jun.

Article in En | MEDLINE | ID: mdl-19269986

Subject(s)

Atrial Fibrillation/metabolism; Natriuretic Peptide, Brain/metabolism; Peptide Fragments/metabolism; Receptor, Angiotensin, Type 1/metabolism; Tachycardia/metabolism; Animals; Atrial Fibrillation/pathology; Coronary Circulation/physiology; Heart Ventricles/metabolism; Heart Ventricles/pathology; Microcirculation/physiology; NADP/metabolism; Oxidative Stress/physiology; Swine; Tachycardia/pathology; Up-Regulation

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Full text: 1 Database: MEDLINE Main subject: Peptide Fragments / Atrial Fibrillation / Tachycardia / Natriuretic Peptide, Brain / Receptor, Angiotensin, Type 1 Type of study: Prognostic_studies Limits: Animals Language: En Year: 2009 Type: Article

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