Srs2 overexpression reveals a helicase-independent role at replication forks that requires diverse cell functions.

León Ortiz, Ana María; Reid, Robert J D; Dittmar, John C; Rothstein, Rodney; Nicolas, Alain

León Ortiz, Ana María; Reid, Robert J D; Dittmar, John C; Rothstein, Rodney; Nicolas, Alain.

Affiliation

León Ortiz AM; Recombination and Genome Instability, Institut Curie Centre de Recherche, CNRS UMR, Université Pierre et Marie Curie, Paris, France. Ana-Maria.Leon@curie.fr

DNA Repair (Amst) ; 10(5): 506-17, 2011 May 05.

Article in En | MEDLINE | ID: mdl-21459050

Subject(s)

DNA Helicases/genetics; DNA Helicases/metabolism; DNA Replication/genetics; Gene Expression/genetics; Saccharomyces cerevisiae Proteins/genetics; Saccharomyces cerevisiae Proteins/metabolism; Saccharomyces cerevisiae/enzymology; Saccharomyces cerevisiae/genetics; Cell Cycle/genetics; Gene Expression Profiling; Gene Expression Regulation, Fungal/genetics; Microbial Viability/genetics; Mutant Proteins/genetics; Mutant Proteins/metabolism; Proliferating Cell Nuclear Antigen/metabolism; Protein Binding; Rad51 Recombinase/metabolism; Saccharomyces cerevisiae/metabolism; Sumoylation/physiology

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Full text: 1 Database: MEDLINE Main subject: Saccharomyces cerevisiae / Gene Expression / DNA Helicases / Saccharomyces cerevisiae Proteins / DNA Replication Language: En Year: 2011 Type: Article

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