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CD39 improves survival in microbial sepsis by attenuating systemic inflammation.
Csóka, Balázs; Németh, Zoltán H; Töro, Gábor; Koscsó, Balázs; Kókai, Endre; Robson, Simon C; Enjyoji, Keiichi; Rolandelli, Rolando H; Erdélyi, Katalin; Pacher, Pál; Haskó, György.
Affiliation
  • Csóka B; Department of Surgery and Center for Immunity and Inflammation, Rutgers New Jersey Medical School, Newark, New Jersey, USA;
  • Németh ZH; Department of Surgery and.
  • Koscsó B; Department of Surgery and Center for Immunity and Inflammation, Rutgers New Jersey Medical School, Newark, New Jersey, USA;
  • Robson SC; Department of Medicine, Gastroenterology and Transplant Institute, Beth Israel Deaconess Medical Center and Harvard Medical School, Boston, Massachusetts, USA; and.
  • Enjyoji K; Department of Medicine, Gastroenterology and Transplant Institute, Beth Israel Deaconess Medical Center and Harvard Medical School, Boston, Massachusetts, USA; and.
  • Erdélyi K; National Institute on Alcohol Abuse and Alcoholism, Bethesda, Maryland, USA.
  • Pacher P; National Institute on Alcohol Abuse and Alcoholism, Bethesda, Maryland, USA.
  • Haskó G; Department of Surgery and Center for Immunity and Inflammation, Rutgers New Jersey Medical School, Newark, New Jersey, USA; haskoge@njms.rutgers.edu.
FASEB J ; 29(1): 25-36, 2015 Jan.
Article in En | MEDLINE | ID: mdl-25318479
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Full text: 1 Database: MEDLINE Main subject: Apyrase / Antigens, CD / Sepsis / Inflammation Limits: Animals / Humans / Male Language: En Year: 2015 Type: Article
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Full text: 1 Database: MEDLINE Main subject: Apyrase / Antigens, CD / Sepsis / Inflammation Limits: Animals / Humans / Male Language: En Year: 2015 Type: Article