Inefficient degradation of cyclin B1 re-activates the spindle checkpoint right after sister chromatid disjunction.

Clijsters, Linda; van Zon, Wouter; Riet, Bas Ter; Voets, Erik; Boekhout, Michiel; Ogink, Janneke; Rumpf-Kienzl, Cornelia; Wolthuis, Rob M F

Clijsters, Linda; van Zon, Wouter; Riet, Bas Ter; Voets, Erik; Boekhout, Michiel; Ogink, Janneke; Rumpf-Kienzl, Cornelia; Wolthuis, Rob M F.

Affiliation

Clijsters L; a Division of Cell Biology I (B5) and Division of Molecular Carcinogenesis (B7); The Netherlands Cancer Institute (NKI-AvL); Amsterdam, The Netherlands.

Cell Cycle ; 13(15): 2370-8, 2014.

Article in En | MEDLINE | ID: mdl-25483188

Subject(s)

Cyclin B1/metabolism; M Phase Cell Cycle Checkpoints/physiology; Proteolysis; Sister Chromatid Exchange/physiology; Aurora Kinase B/metabolism; CDC2 Protein Kinase; Cdc20 Proteins/metabolism; Cell Line, Tumor; Cyclin B1/genetics; Cyclin-Dependent Kinases/metabolism; Humans; Lysine/metabolism; Merozoite Surface Protein 1/metabolism; Mutation

Key words

APC/C; Cdc20; Cdk1; anaphase; cyclin B1; metaphase; spindle checkpoint

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Full text: 1 Database: MEDLINE Main subject: Sister Chromatid Exchange / Cyclin B1 / Proteolysis / M Phase Cell Cycle Checkpoints Limits: Humans Language: En Year: 2014 Type: Article

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