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Decreased Satellite Cell Number and Function in Humans and Mice With Type 1 Diabetes Is the Result of Altered Notch Signaling.
D'Souza, Donna M; Zhou, Sarah; Rebalka, Irena A; MacDonald, Blair; Moradi, Jasmin; Krause, Matthew P; Al-Sajee, Dhuha; Punthakee, Zubin; Tarnopolsky, Mark A; Hawke, Thomas J.
Affiliation
  • D'Souza DM; Department of Pathology & Molecular Medicine, McMaster University, Hamilton, Ontario, Canada.
  • Zhou S; Department of Pathology & Molecular Medicine, McMaster University, Hamilton, Ontario, Canada.
  • Rebalka IA; Department of Pathology & Molecular Medicine, McMaster University, Hamilton, Ontario, Canada.
  • MacDonald B; Department of Pathology & Molecular Medicine, McMaster University, Hamilton, Ontario, Canada.
  • Moradi J; Department of Pathology & Molecular Medicine, McMaster University, Hamilton, Ontario, Canada.
  • Krause MP; Department of Kinesiology, University of Windsor, Windsor, Ontario, Canada.
  • Al-Sajee D; Department of Pathology & Molecular Medicine, McMaster University, Hamilton, Ontario, Canada.
  • Punthakee Z; Department of Pediatrics, McMaster University, Hamilton, Ontario, Canada.
  • Tarnopolsky MA; Department of Pediatrics, McMaster University, Hamilton, Ontario, Canada.
  • Hawke TJ; Department of Pathology & Molecular Medicine, McMaster University, Hamilton, Ontario, Canada hawke@mcmaster.ca.
Diabetes ; 65(10): 3053-61, 2016 10.
Article in En | MEDLINE | ID: mdl-27335233

Full text: 1 Database: MEDLINE Main subject: Satellite Cells, Skeletal Muscle / Receptors, Notch Limits: Adolescent / Adult / Animals / Humans / Male Language: En Year: 2016 Type: Article

Full text: 1 Database: MEDLINE Main subject: Satellite Cells, Skeletal Muscle / Receptors, Notch Limits: Adolescent / Adult / Animals / Humans / Male Language: En Year: 2016 Type: Article