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ESCRTing Necroptosis.
Guo, Hongyan; Kaiser, William J.
Affiliation
  • Guo H; Department of Microbiology, Immunology, and Molecular Genetics, University of Texas Heath San Antonio, 7703 Floyd Curl Drive, San Antonio, TX 78229, USA.
  • Kaiser WJ; Department of Microbiology, Immunology, and Molecular Genetics, University of Texas Heath San Antonio, 7703 Floyd Curl Drive, San Antonio, TX 78229, USA. Electronic address: kaiserw@uthscsa.edu.
Cell ; 169(2): 186-187, 2017 04 06.
Article in En | MEDLINE | ID: mdl-28388403
Necroptosis is a highly inflammatory form of programmed cell death that results from MLKL-mediated disruption of the cell membrane. In this issue of Cell, Gong et al. challenge the notion that MLKL activation is a point of no return by identifying mechanisms to counterbalance necroptosis, sustain plasma membrane integrity, and prolong cell viability.
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Full text: 1 Database: MEDLINE Main subject: Protein Kinases / Receptor-Interacting Protein Serine-Threonine Kinases Language: En Year: 2017 Type: Article
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Full text: 1 Database: MEDLINE Main subject: Protein Kinases / Receptor-Interacting Protein Serine-Threonine Kinases Language: En Year: 2017 Type: Article