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MAFB prevents excess inflammation after ischemic stroke by accelerating clearance of damage signals through MSR1.
Shichita, Takashi; Ito, Minako; Morita, Rimpei; Komai, Kyoko; Noguchi, Yoshiko; Ooboshi, Hiroaki; Koshida, Ryusuke; Takahashi, Satoru; Kodama, Tatsuhiko; Yoshimura, Akihiko.
Affiliation
  • Shichita T; Department of Microbiology and Immunology, School of Medicine, Keio University, Tokyo, Japan.
  • Ito M; Core Research for Evolutional Science and Technology (CREST), Japan Agency for Medical Research and Development (AMED), Tokyo, Japan.
  • Morita R; Precursory Research for Embryonic Science and Technology (PRESTO), Japan Science and Technology Agency, Tokyo, Japan.
  • Komai K; Stroke Renaissance Project, Tokyo Metropolitan Institute of Medical Science, Tokyo, Japan.
  • Noguchi Y; Department of Medicine and Clinical Science, Graduate School of Medical Sciences, Kyushu University, Fukuoka, Japan.
  • Ooboshi H; Department of Microbiology and Immunology, School of Medicine, Keio University, Tokyo, Japan.
  • Koshida R; Core Research for Evolutional Science and Technology (CREST), Japan Agency for Medical Research and Development (AMED), Tokyo, Japan.
  • Takahashi S; Department of Microbiology and Immunology, School of Medicine, Keio University, Tokyo, Japan.
  • Kodama T; Core Research for Evolutional Science and Technology (CREST), Japan Agency for Medical Research and Development (AMED), Tokyo, Japan.
  • Yoshimura A; Department of Microbiology and Immunology, School of Medicine, Keio University, Tokyo, Japan.
Nat Med ; 23(6): 723-732, 2017 Jun.
Article in En | MEDLINE | ID: mdl-28394332
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Full text: 1 Database: MEDLINE Main subject: Brain / Receptors, Immunologic / Infarction, Middle Cerebral Artery / Myeloid Cells / MafB Transcription Factor / Scavenger Receptors, Class A / Alarmins Type of study: Prognostic_studies Limits: Animals Language: En Year: 2017 Type: Article
Fulltext
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Full text: 1 Database: MEDLINE Main subject: Brain / Receptors, Immunologic / Infarction, Middle Cerebral Artery / Myeloid Cells / MafB Transcription Factor / Scavenger Receptors, Class A / Alarmins Type of study: Prognostic_studies Limits: Animals Language: En Year: 2017 Type: Article