Your browser doesn't support javascript.
loading
Protein kinase Cα gain-of-function variant in Alzheimer's disease displays enhanced catalysis by a mechanism that evades down-regulation.
Callender, Julia A; Yang, Yimin; Lordén, Gema; Stephenson, Natalie L; Jones, Alexander C; Brognard, John; Newton, Alexandra C.
Affiliation
  • Callender JA; Department of Pharmacology, University of California, San Diego, La Jolla, CA 92093.
  • Yang Y; Biomedical Sciences Graduate Program, University of California, San Diego, La Jolla, CA 92093.
  • Lordén G; Department of Pharmacology, University of California, San Diego, La Jolla, CA 92093.
  • Stephenson NL; Department of Pharmacology, University of California, San Diego, La Jolla, CA 92093.
  • Jones AC; Cancer Research UK Manchester Institute, The University of Manchester, Manchester 4BX M20, United Kingdom.
  • Brognard J; Biomedical Sciences Graduate Program, University of California, San Diego, La Jolla, CA 92093.
  • Newton AC; Cancer Research UK Manchester Institute, The University of Manchester, Manchester 4BX M20, United Kingdom.
Proc Natl Acad Sci U S A ; 115(24): E5497-E5505, 2018 06 12.
Article in En | MEDLINE | ID: mdl-29844158
Subject(s)
Key words
Fulltext
Print
XML
PubMed Links
Search on Google

Full text: 1 Database: MEDLINE Main subject: Down-Regulation / Protein Kinase C-alpha / Alzheimer Disease / Gain of Function Mutation Limits: Animals / Humans Language: En Year: 2018 Type: Article
Fulltext
Print
XML
PubMed Links
Search on Google

Full text: 1 Database: MEDLINE Main subject: Down-Regulation / Protein Kinase C-alpha / Alzheimer Disease / Gain of Function Mutation Limits: Animals / Humans Language: En Year: 2018 Type: Article