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Hmga2 is dispensable for pancreatic cancer development, metastasis, and therapy resistance.
Chiou, Shin-Heng; Dorsch, Madeleine; Kusch, Eva; Naranjo, Santiago; Kozak, Margaret M; Koong, Albert C; Winslow, Monte M; Grüner, Barbara M.
Affiliation
  • Chiou SH; Department of Genetics, Stanford University School of Medicine, Stanford, CA, USA.
  • Dorsch M; Department of Medical Oncology, West German Cancer Center, University Hospital Essen, University Duisburg-Essen, Essen, Germany.
  • Kusch E; Department of Medical Oncology, West German Cancer Center, University Hospital Essen, University Duisburg-Essen, Essen, Germany.
  • Naranjo S; Department of Genetics, Stanford University School of Medicine, Stanford, CA, USA.
  • Kozak MM; Department of Radiation Oncology, Stanford University School of Medicine, Stanford, CA, USA.
  • Koong AC; Department of Radiation Oncology, Stanford University School of Medicine, Stanford, CA, USA.
  • Winslow MM; Department of Radiation Oncology, MD Anderson Cancer Center, The University of Texas, Houston, TX, USA.
  • Grüner BM; Department of Genetics, Stanford University School of Medicine, Stanford, CA, USA. mwinslow@stanford.edu.
Sci Rep ; 8(1): 14008, 2018 09 18.
Article in En | MEDLINE | ID: mdl-30228296
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Full text: 1 Database: MEDLINE Main subject: Pancreatic Neoplasms / Adenocarcinoma / Drug Resistance, Neoplasm / Carcinoma, Pancreatic Ductal / HMGA2 Protein / Deoxycytidine / Cell Proliferation Type of study: Prognostic_studies Limits: Animals / Humans Language: En Year: 2018 Type: Article
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Full text: 1 Database: MEDLINE Main subject: Pancreatic Neoplasms / Adenocarcinoma / Drug Resistance, Neoplasm / Carcinoma, Pancreatic Ductal / HMGA2 Protein / Deoxycytidine / Cell Proliferation Type of study: Prognostic_studies Limits: Animals / Humans Language: En Year: 2018 Type: Article