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Inactive variants of death receptor p75NTR reduce Alzheimer's neuropathology by interfering with APP internalization.
Yi, Chenju; Goh, Ket Yin; Wong, Lik-Wei; Ramanujan, Ajeena; Tanaka, Kazuhiro; Sajikumar, Sreedharan; Ibáñez, Carlos F.
Affiliation
  • Yi C; Department of Physiology, National University of Singapore, Singapore City, Singapore.
  • Goh KY; Life Sciences Institute, National University of Singapore, Singapore City, Singapore.
  • Wong LW; Department of Physiology, National University of Singapore, Singapore City, Singapore.
  • Ramanujan A; Life Sciences Institute, National University of Singapore, Singapore City, Singapore.
  • Tanaka K; Department of Physiology, National University of Singapore, Singapore City, Singapore.
  • Sajikumar S; Life Sciences Institute, National University of Singapore, Singapore City, Singapore.
  • Ibáñez CF; Department of Physiology, National University of Singapore, Singapore City, Singapore.
EMBO J ; 40(2): e104450, 2021 01 15.
Article in En | MEDLINE | ID: mdl-33258176
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Full text: 1 Database: MEDLINE Main subject: Amyloid beta-Protein Precursor / Receptors, Nerve Growth Factor / Protein Transport / Alzheimer Disease / Nerve Tissue Proteins Type of study: Prognostic_studies Limits: Animals / Humans Language: En Year: 2021 Type: Article
Fulltext
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PubMed Links
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Full text: 1 Database: MEDLINE Main subject: Amyloid beta-Protein Precursor / Receptors, Nerve Growth Factor / Protein Transport / Alzheimer Disease / Nerve Tissue Proteins Type of study: Prognostic_studies Limits: Animals / Humans Language: En Year: 2021 Type: Article