Oncogenic collaboration of the cyclin D1 (PRAD1, bcl-1) gene with a mutated p53 and an activated ras oncogene in neoplastic transformation.

Uchimaru, K; Endo, K; Fujinuma, H; Zukerberg, L; Arnold, A; Motokura, T

Uchimaru, K; Endo, K; Fujinuma, H; Zukerberg, L; Arnold, A; Motokura, T.

Affiliation

Uchimaru K; Fourth Department of Internal Medicine, University of Tokyo, School of Medicine, Japan.

Jpn J Cancer Res ; 87(5): 459-65, 1996 May.

Article in En | MEDLINE | ID: mdl-8641982

Subject(s)

Cell Transformation, Neoplastic/genetics; Cyclins/genetics; Genes, p53/physiology; Genes, ras/physiology; Oncogene Proteins/genetics; Oncogenes/physiology; Animals; Cell Transformation, Neoplastic/metabolism; Cyclin D1; Cyclins/metabolism; Cyclins/physiology; Fetus; Fibroblasts/metabolism; Genetic Vectors; Mice; Mice, Nude; Oncogene Proteins/metabolism; Oncogene Proteins/physiology; Rats; Rats, Wistar; Transfection

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Full text: 1 Database: MEDLINE Main subject: Oncogenes / Cell Transformation, Neoplastic / Genes, p53 / Genes, ras / Oncogene Proteins / Cyclins Limits: Animals Language: En Year: 1996 Type: Article

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