Glucocorticoid receptor-mediated cell cycle arrest is achieved through distinct cell-specific transcriptional regulatory mechanisms.

Rogatsky, I; Trowbridge, J M; Garabedian, M J

Rogatsky, I; Trowbridge, J M; Garabedian, M J.

Affiliation

Rogatsky I; Department of Microbiology and The Kaplan Cancer Center, New York University Medical Center, New York 10016, USA.

Mol Cell Biol ; 17(6): 3181-93, 1997 Jun.

Article in En | MEDLINE | ID: mdl-9154817

Subject(s)

Cell Cycle Proteins; Cell Cycle/genetics; Proto-Oncogene Proteins; Receptors, Glucocorticoid/metabolism; Transcription, Genetic; Tumor Suppressor Proteins; Animals; Binding Sites; Cell Division; Cyclin D3; Cyclin-Dependent Kinase 4; Cyclin-Dependent Kinase 6; Cyclin-Dependent Kinase Inhibitor p21; Cyclin-Dependent Kinase Inhibitor p27; Cyclin-Dependent Kinases/metabolism; Cyclins/metabolism; DNA/metabolism; Enzyme Inhibitors/metabolism; Humans; Mice; Microtubule-Associated Proteins/metabolism; Protein Serine-Threonine Kinases/metabolism; Rats; Retinoblastoma Protein/metabolism; Tumor Cells, Cultured

Fulltext

XML

PubMed Links

Search on Google

Full text: 1 Database: MEDLINE Main subject: Transcription, Genetic / Receptors, Glucocorticoid / Cell Cycle / Proto-Oncogene Proteins / Cell Cycle Proteins / Tumor Suppressor Proteins Limits: Animals / Humans Language: En Year: 1997 Type: Article

Fulltext

XML

PubMed Links

Search on Google