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PGE2-induced IDO1 inhibits the capacity of fully mature DCs to elicit an in vitro antileukemic immune response.
Trabanelli, Sara; Lecciso, Mariangela; Salvestrini, Valentina; Cavo, Michele; Ocadlíková, Darina; Lemoli, Roberto M; Curti, Antonio.
Afiliación
  • Trabanelli S; Department of Specialistic, Diagnostic and Experimental Medicine, Institute of Hematology "L. & A. Seràgnoli", University of Bologna, 40138 Bologna, Italy ; Ludwig Center for Cancer Research of the University of Lausanne, 1011 Lausanne, Switzerland.
  • Lecciso M; Department of Specialistic, Diagnostic and Experimental Medicine, Institute of Hematology "L. & A. Seràgnoli", University of Bologna, 40138 Bologna, Italy.
  • Salvestrini V; Department of Specialistic, Diagnostic and Experimental Medicine, Institute of Hematology "L. & A. Seràgnoli", University of Bologna, 40138 Bologna, Italy.
  • Cavo M; Department of Specialistic, Diagnostic and Experimental Medicine, Institute of Hematology "L. & A. Seràgnoli", University of Bologna, 40138 Bologna, Italy.
  • Ocadlíková D; Department of Specialistic, Diagnostic and Experimental Medicine, Institute of Hematology "L. & A. Seràgnoli", University of Bologna, 40138 Bologna, Italy.
  • Lemoli RM; Department of Internal Medicine (DiMI), University of Genoa and IRCCS Azienda Ospedaliera Universitaria S. Martino-IST, 16132 Genoa, Italy.
  • Curti A; Department of Specialistic, Diagnostic and Experimental Medicine, Institute of Hematology "L. & A. Seràgnoli", University of Bologna, 40138 Bologna, Italy.
J Immunol Res ; 2015: 253191, 2015.
Article en En | MEDLINE | ID: mdl-25815345

Texto completo: 1 Banco de datos: MEDLINE Asunto principal: Células Dendríticas / Dinoprostona / Citotoxicidad Inmunológica / Indolamina-Pirrol 2,3,-Dioxigenasa Límite: Humans Idioma: En Año: 2015 Tipo del documento: Article

Texto completo: 1 Banco de datos: MEDLINE Asunto principal: Células Dendríticas / Dinoprostona / Citotoxicidad Inmunológica / Indolamina-Pirrol 2,3,-Dioxigenasa Límite: Humans Idioma: En Año: 2015 Tipo del documento: Article