RESUMEN
The radon ((222)Rn) activity concentration in 15 dwellings in the Planej village and 10 dwellings in the Gorozhup village has been measured with the aim to complement the national radon survey and to compare the results of two different measurement techniques. The radon concentration has been measured in winter and spring using alpha scintillation cells and in winter, spring and summer by exposing solid-state nuclear track detectors. Both methods gave similar results. Radon concentrations in both villages were similar, ranging from 82 to 432 Bq m(-3); the value of 400 Bq m(-3) was exceeded only in two dwellings. The resulting annual effective doses ranged from 1.78 to 6.40 mSv, with the average values of 3.28 mSv in the Planej village and 3.87 mSv in the Gorozhup village.
Asunto(s)
Monitoreo de Radiación/métodos , Radón/análisis , Uranio/análisis , Contaminantes Radiactivos del Aire/análisis , Contaminación del Aire Interior/análisis , Geografía , Vivienda , Kosovo , Dosis de Radiación , Estaciones del AñoRESUMEN
Approximately 30 patients with familial hypomagnesemia-hypercalciuria have been reported. We describe an 8-year-old girl with cardinal findings of familial hypomagnesemia-hypercalciuria (hypomagnesemia, hypermagnesiuria, hypercalciuria, renal insufficiency, hyperuricemia, elevated serum parathormone, hyposthenuria and nephrocalcinosis), who received combination therapy consisting of magnesium salts, thiazide diuretic and potassium supplementation. At the 4-year follow-up investigation under this treatment, the patient was found to have cerebral pseudotumor (increased intracranial pressure with normal or small ventricles on neuroimaging, no evidence of an intracranial mass and normal cerebrospinal fluid composition) with papilledema and visual field defects. Thiazide therapy was terminated and the cerebral pseudotumor disappeared. The authors hypothesize that cerebral pseudotumor in this patient was related to severe hypocalcemia, as a consequence of profound hypomagnesemia induced by protracted thiazide treatment. To our knowledge, this is the first report of a child with familial hypomagnesemia-hypercalciuria who developed pseudotumor cerebri after thiazide therapy.