[Effect of nootropil on oxidative phosphorylation in the brain of rats with ischemic myocardial necrosis]. / Vliianie nootropila na okislitel'noe fosforilirovanie v mozge krys s ishemicheskim nekrozom miokarda.

In experiments on rats the formation of ischemic myocardial necrosis was shown to be followed by dissociation of the processes of oxidation and phosphorylation in mitochondria of the cerebral hemispheres. Administration of nootropil to the animals in the acute period prevented the occurrence of these disturbances.

[Effect of nootropil on the conditioned reflex activity in acute myocardial infarct]. / Vliianie nootropila na uslovnoreflektornuiu deiatel'nost' pri ostrom infarkte miokarda.

At the acute stage of myocardial infarction the higher functions of the brain such as fixation and reproduction of temporary connections were shown to be markedly depressed. The use of nootropil in experimental myocardial infarction was followed by the improvement of conditioned-reflex activity.

[Alteration of Ca2+, Mg2+-ATPase activity from rat hypothalamus and myocardium in development of the neurotic state]. / Izmeneniia aktivnosti Ca, Mg-zavisimoi ATf-azy v gipotalamuse i miokarde pri formirovanii nevroticheskogo sostoianiia u krys.

Simultaneous decrease of Ca2+, Mg2+-ATPase activity occurred in rat hypothalamus and myocardium during experimental neurosis. The decrease of the enzymatic activity was more distinct in hypothalamus than in myocardium. In hypothalamus the decreased enzymatic activity maintained within a week after completion of the excitation of the animals.