Dam construction reshapes sedimentary pollutant distribution along the Yangtze river by regulating sediment composition.

Dam construction has far-reaching impacts on pollutant accumulation and the pollutant-induced quality of aquatic environments. Nonetheless, its large-scale effects on pollutant distribution in sediments, which greatly contribute to the environmental impacts of coexisting pollutants, remain poorly understood. We collected sediments from the Yangtze River during the dry and normal seasons (with 'normal' defined in terms of precipitation level), and examined how dam construction alters the spatial trajectories of both inorganic and organic pollutants in the sediments. Sediment composition exhibited linear variation from the upper to the lower reaches, with clay and silt particles dominating the sediment in the Three Gorges Reservoir and sand particles dominating in the middle-lower reaches. Accordingly, upstream of the Three Gorges Dam (TGD), sedimentary carbon, nitrogen, phosphorus, heavy metal, polycyclic aromatic hydrocarbons (PAHs), and oxygenated PAHs (OPAHs) contents increased toward the TGD owing to its regulation of the spatial variation in sediment particle size. The TGD caused upstream sedimentary accumulation of pollutants to be higher nearer to the TGD than in the upper reaches by 17%-129% for carbon, nitrogen, and phosphorus, 7%-51% for heavy metals, 30% for PAHs, and 140% for OPAHs. Pollutant content was sharply lower below the TGD, by 0.58-11.15 times for carbon, nitrogen, and phosphorus, 0.1-2.6 times for heavy metals, 1.7 times for PAHs, and 5.6 times for OPAHs. Upstream of the TGD, levels of NH4+-N, the main form of N in the interstitial water of the Yangtze River, increased lineary toward the TGD, whereas those of NO3--N and NO2--N decreased. Sedimentary organic matter source contributions were consistent along the Yangtze River, being on an average 46% for C3 plants and 28% for soil organic substances, further confirming the dam's regulatory effect on pollutants. These findings provide a foundation for future assessments of the environmental impact of dam-induced river fragmentation and hydrological alterations, and for developing advanced watershed pollutant management strategies.

Nutrients removal and recovery from anaerobically digested swine wastewater by struvite crystallization without chemical additions.

Anaerobically digested swine wastewater contains high concentrations of phosphorus (P) and nitrogen (N). A pilot-scale experiment was carried out for nutrients removal and recovery from anaerobically digested swine wastewater by struvite crystallization. In the pilot plant, a sequencing batch reactor (SBR) and a continuous-flow reactor with struvite accumulation devices were designed and employed. The wastewater pH value was increased by CO(2) stripping, and the struvite crystallization process was performed without alkali and Mg(2+) additions. Results of the long-term operation of the system showed that, both reactors provided up to 85% P removal and recovery over wide ranges of aeration times (1.0-4.0 h), hydraulic retention times (HRT) (6.0-15.0 h) and temperatures (0-29.5°C) for an extended period of 247 d, in which approximate 30% of P was recovered by the struvite accumulation devices. However, 40-90% of NH(4)(+)-N removed was through air stripping instead of being immobilized in the recovered solids. The recovered products were detected and analyzed by scanning electron microscope (SEM), X-ray diffraction (XRD) and chemical methods, which were proved to be struvite with purity of more than 90%. This work demonstrated the feasibility and effects of nutrients removal and recovery from anaerobically digested swine wastewater by struvite crystallization without chemical additions.

Modeling the crystallization of magnesium ammonium phosphate for phosphorus recovery.

The crystallization of magnesium ammonium phosphate (MAP) is one of the main processes for recovering P and N from wastewater. Chemically defined solution systems were designed; the saturation indices (SIs) of the solution systems with respect to MAP were derived by using a geochemical aqueous model Program, PHREEQC 2.11; the effects of the solution conditions were evaluated using thermodynamic theories. The concentrations of P and Mg in the tested solutions were 10-600 mg l(-1) and 24-720 mg l(-1), respectively, the molar ratios of N/P and pH values of the solutions varied in the ranges of 1-40 and 6.0-12.0, respectively. The temperature of all the tests was set at 25 degrees C. The test results show that the SI value of MAP is the logarithmic functions of the concentrations of P, ammonium-N and Mg, and increases with the increase of the concentration of each element. The SI value of MAP is a polynomial function of pH value of the solution, and the optimum pH value for the crystallization of MAP is 9.0 but increases slightly with the increase of the N/P. Moreover, the SI value of MAP is a power law function of the ionic strength of solutions but decreases with its increase. The adjustment of the Mg concentration and the control of solution pH are two effective methods for the control of the crystallization of MAP. The results obtained from the research can be used to guide the design and control of MAP crystallization process for the removal and recovery of P.

The predominant role of brain angiotensinogen and angiotensin in environmentally induced hypertension.

Rats exposed chronically to a cold environment (5 degrees C/4 degrees F) develop hypertension. This cold-induced hypertension (CIH) is a non-genetic, non-pharmacological, non-surgical model of environmentally induced hypertension in rats. The renin-angiotensin system (RAS) appears to play a role in both initiating and/or maintaining the high blood pressure in CIH. The goal of the present study was to evaluate the role of central and peripheral circulating RAS components, angiotensinogen (AGT), angiotensin-converting enzyme (ACE) and angiotensin (Ang) II, in CIH. Seventy-two Sprague-Dawley adult male rats were used. Thirty-six rats were kept in cold room at 5 degrees C while the other 36 were at 24 degrees C as controls for 5 weeks. Systolic blood pressure (SBP) was recorded by tail cuff. The SBP was increased in rats exposed to cold within 1 week, and this increase was significant for the next 2-5 weeks of the cold exposure (p<0.01). Three subgroups of the cold-treated and control rats (n=12) were sacrificed at 1, 3 and 5 weeks. The brain and liver were removed and plasma was saved. The AGT mRNA significantly increased in the hypothalamus and liver in cold-treated rats from the first week of exposure to cold, and was maintained throughout the time of exposure to cold (n=4, p<0.01). The AGT protein levels in the brain, liver and plasma did not differ significantly between cold-treated and control rats (p>0.05, n=4). The hypothalamic Ang II levels were significantly increased, whereas plasma Ang II levels significantly decreased, in the rats of 5 weeks of cold exposure (n=8, p<0.05). Plasma ACE significantly increased in the rats of 1 week of cold exposure (p<0.05, n=12). The results show differential regulation of RAS components, AGT, ACE and Ang II, between brain and periphery in cold-exposed rats. We conclude that the exposure to low temperature initially increases plasma RAS but with continuous exposure to cold, the brain RAS maintains the hypertension, probably by sustained sympathetic activation, which would provide increased metabolism but also vasoconstriction leading to hypertension.